Preparation of non-sintered lightweight aggregates through co-mechanochemical treatment of oil-contaminated drill cuttings, circulation fluidized bed combustion fly ash, and quicklime.

This work investigates the feasibility of co-mechanochemical treatment of oil-contaminated drill cuttings (OCDC), circulation fluidized bed combustion (CFBC) fly ash, and quicklime to prepare non-sintered lightweight aggregates (NSLWAs). The NSLWAs with high cylinder compressive strength and low water absorption could be obtained under the condition of optimal water addition and appropriate steam-curing temperature, as well as steam-curing time. Co-mechanochemical treatment could enhance the pozzolanic reactivity of CFBC fly ash effectively, which is beneficial to the strength development of NSLWAs. Moreover, co-mechanochemical treatment also can degrade the petroleum hydrocarbon of OCDC, greatly reducing the leaching concentrations of total petroleum hydrocarbons (TPH) of NSLWAs. The final leaching concentrations of TPH are much lower than the requirements of Chinese National Standard GB 31571-2015. Graphical abstract.

Clinical chorioamnionitis at term: the amniotic fluid fatty acyl lipidome.

Clinical chorioamnionitis at term (TCC) is the most common obstetrical infliction diagnosed in labor and delivery units worldwide and is associated with a substantial increase in maternal and neonatal morbidity and mortality. This obstetrical complication is a heterogeneous condition, as only half of patients have detectable microorganisms in the amniotic cavity. Because bioactive lipids play a key role in the initiation and resolution of an inflammatory response, we aimed to characterize the amniotic fluid lipidome in patients with TCC. We studied the amniotic fluid of patients in the following groups: 1) spontaneous labor at term without clinical chorioamnionitis (TLB) and 2) spontaneous labor at term with clinical chorioamnionitis (TCC). The TCC group was subdivided into a) those with microbial invasion of the amniotic cavity (TCC-MIAC) and b) those without microbial invasion of the amniotic cavity (TCC-noMIAC). The amniotic fluid concentration of proinflammatory lipid mediators did not differ between patients in TLB with TCC. In contrast, concentration of lipids with anti-inflammatory/proresolution properties was significantly lower in all patients with TCC than in those with TLB. These results suggest that while proinflammatory lipid mediators are involved in infection-driven intra-amniotic inflammation, a relative deficiency of anti-inflammatory/proresolution lipid mediator biosynthesis is a characteristic of TCC.