Relação entre a progressão do escore do functional movement screen e lesão em atletas futebol / Relationship between progression of functional movement screen test and injury in athletes of football

O futebol é uma modalidade de grande exigência física com movimentos de potência e velocidade com mudança de direção. Devido à alta intensidade, ocorre uma maior predisposição às lesões. O Functional Movement Screen (FMS) é um modelo de avaliação para identificar assimetrias e desequilíbrios musculoesqueléticos. O objetivo deste trabalho foi comparar os efeitos de do is m eses de pré-temporada na progressão do escore do teste FMS e sua relação com as lesões ao longo da competição. Participaram deste estudo 28 atletas profissionais de futebol, com idade média de 25,1 ± 6,5. Foi realizado o FMS antes e após a pré-temporada e durante o período competitivo foi verificada a ocorrência de lesões. Houve uma melhora (p<0,01) dos escores do FMS da avaliação pré (15,61 ± 1,39) para a avaliação pós pré-temporada (17,29 ± 1,24) entre todo o elenco, porém, quando considerado o tamanho do efeito, os atletas que não se lesionaram tiveram um efeito maior na progressão do escore (d=1,17) do que os que se lesionaram (d=0,35). Conclui-se que os atletas que não se lesionaram durante a competição, foram os que mais progrediram no FMS durante a pré-temporada, sendo, provavelmente, a progressão do escore do s atletas mais interessante do que o atendimento ao ponto de corte sugerido pela literatura...(AU)

Football is a modality that demands high physical fitness, with intense movements and rapid direction changing. Due to the high intensity demanded, athletes are more susceptible t o in juries. The Functional Movement Screen (FMS) test it is a method to evaluate asymmetries and muscle im balance. The objective was to compared the effects of two months of preseason, on the score of the FMS t est and their relationship with the injuries resulting from the competitive period. Participated o f th is study 28 professional football players (25.1 ± 6.5 years). FMS was performed before and after the p reseason and during the competitive period the occurrence of injuries was verified. There was a significant improvement of the FMS score after the preseason (p<0.01) for the all athletes. When considering the size effect, athletes who were not injured during the competitive period had a greater effect (d=1.17) than those who injured (d=0.35). The athletes who progressed the most during the evaluation were the athletes who were exempted from injury during the competitive period. Perhaps the progression of the athlete's score was more interesting than the attendance to the cut-off point suggested by the literature...(AU)

Hepatic heme synthesis in a new model of experimental hemochromatosis: studies in rats fed finely divided elemental iron.

Rats fed chow containing finely divided elemental iron (from carbonyl-iron) develop hepatic iron overload resembling human hereditary hemochromatosis in that deposition of iron is primarily in periportal hepatocytes and with hepatic iron concentrations sufficiently high to be associated in the human disease with hepatic fibrosis or cirrhosis. In recent studies using this model, we reported changes in hepatic hemoproteins and heme oxygenase, the rate-controlling enzyme of heme breakdown. We now report effects of iron-loading on three enzymes of heme synthesis: 5-aminolevulinate synthase; the first and rate-controlling enzyme of the pathway, 5-aminolevulinate dehydrase (or porphobilinogen synthase), and uroporphyrinogen decarboxylase, the activity of which is decreased in porphyria cutanea tarda, a liver disease in which iron is known to play an important but still poorly understood role. Of the three enzymes, only activity of the dehydrase was altered by iron-loading: it was decreased significantly as early as 1 week after starting iron feeding, and with marked iron overload was 30 to 32% of control values. The degree of decrease was inversely related (r = -0.77 to -0.88) to the degree of iron overload and was partially reversed within 1 to 3 days when feeding of the iron-supplemented diet was stopped. The decrease in dehydrase activity was not attributable to lack of reduced glutathione or other disulfide-reducing agents or to zinc deficiency; nor was evidence found for inhibition by iron compounds or other possible inhibitors present in iron-loaded livers.(ABSTRACT TRUNCATED AT 250 WORDS)