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Mol Pharm ; 17(3): 757-768, 2020 03 02.
Artigo em Inglês | MEDLINE | ID: mdl-32011888

RESUMO

Salmon calcitonin (sCT) is a potent calcium-regulating peptide hormone and widely applied for the treatment of some bone diseases clinically. However, the therapeutic usefulness of sCT is hindered by the frequent injection required, owing to its short plasma half-life and therapeutic need for a high dose. Oral delivery is a popular modality of administration for patients because of its convenience to self-administration and high patient compliance, while orally administered sCT remains a great challenge currently due to the existence of multiple barriers in the gastrointestinal (GI) tract. Here, we introduced an orally targeted delivery system to increase the transport of sCT across the intestine through both the paracellular permeation route and the bile acid pathway. In this system, sCT-based glycol chitosan-taurocholic acid conjugate (GC-T)/dextran sulfate (DS) ternary nanocomplexes (NC-T) were produced by a flash nanocomplexation (FNC) process in a kinetically controlled mode. The optimized NC-T exhibited well-controlled properties with a uniform and sub-60 nm hydrodynamic diameter, high batch-to-batch reproducibility, good physical or chemical stability, as well as sustained drug release behaviors. The studies revealed that NC-T could effectively improve the intestinal uptake and permeability, owing to its surface functionalization with the taurocholic acid ligand. In the rat model, orally administered NC-T showed an obvious hypocalcemia effect and a relative oral bioavailability of 10.9%. An in vivo assay also demonstrated that NC-T induced no observable side effect after long-term oral administration. As a result, the orally targeted nanocomplex might be a promising candidate for improving the oral transport of therapeutic peptides.


Assuntos
Calcitonina/administração & dosagem , Hormônios e Agentes Reguladores de Cálcio/administração & dosagem , Sistemas de Liberação de Medicamentos/métodos , Absorção Intestinal/efeitos dos fármacos , Nanocompostos/química , Administração Oral , Animais , Disponibilidade Biológica , Transporte Biológico , Células CACO-2/efeitos dos fármacos , Células CACO-2/metabolismo , Calcitonina/efeitos adversos , Calcitonina/sangue , Calcitonina/farmacocinética , Cálcio/sangue , Hormônios e Agentes Reguladores de Cálcio/efeitos adversos , Hormônios e Agentes Reguladores de Cálcio/sangue , Hormônios e Agentes Reguladores de Cálcio/farmacocinética , Quitosana/química , Sulfato de Dextrana/química , Liberação Controlada de Fármacos , Estabilidade de Medicamentos , Meia-Vida , Humanos , Hipocalcemia/induzido quimicamente , Injeções Subcutâneas , Masculino , Ratos , Ratos Sprague-Dawley , Ácido Taurocólico/química
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