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Pharmacol Res ; 46(5): 383-7, 2002 Nov.
Artigo em Inglês | MEDLINE | ID: mdl-12419641

RESUMO

The present study was aimed at investigating whether the regulation of vascular renin-angiotensin and endothelin (ET) systems is altered by a chronic blockade of nitric oxide (NO) synthesis. Male Sprague-Dawley rats were supplemented with N(G)-nitro-L-arginine methyl ester (L-NAME, 100mgl(-1)) in drinking water for 4 weeks to inhibit the endogenous synthesis of NO. The mRNA expressions of renin, angiotensin converting enzyme (ACE), type-1 angiotensin II receptor (AT1R), ET-1, type-A ET receptor (ET(A)), and neutral endopeptidase (NEP) were determined in the thoracic aorta by reverse transcription-polymerase chain reaction. The treatment with L-NAME significantly increased the blood pressure, while it decreased the tissue levels of nitrite/nitrate. The mRNA expression of renin, ACE, and AT1R was increased in the aorta. The protein expression of AT1R assessed by Western blot analysis was also increased. The expression of ET-1 and ET(A) mRNA was increased, whereas that of NEP mRNA decreased. The increased expression of renin-angiotensin and ET system genes and the decreased expression of NEP may in part be causally related with the development of hypertension induced by a chronic blockade of NO synthesis.


Assuntos
Endotelina-1/genética , Músculo Liso Vascular/metabolismo , Óxido Nítrico/antagonistas & inibidores , Sistema Renina-Angiotensina/genética , Animais , Aorta Torácica/metabolismo , Pressão Sanguínea/efeitos dos fármacos , Endotelina-1/biossíntese , Expressão Gênica/genética , Masculino , NG-Nitroarginina Metil Éster/farmacologia , RNA Mensageiro/análise , Ratos , Ratos Sprague-Dawley , Receptores de Endotelina/biossíntese , Receptores de Endotelina/genética , Reação em Cadeia da Polimerase Via Transcriptase Reversa , Regulação para Cima
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