Injuries related to sauna bathing.

PURPOSE: Awareness of well-being and health issues have contributed to increased popularity of sauna bathing. However, little is known about potential risks and injuries. The aim of this study was to identify the causes for injuries, the affected body regions and to define recommendations for prevention. METHODS: A retrospective chart data analysis was conducted among patients treated for an injury related to sauna bathing at the local trauma centre of the Medical University of Innsbruck between January 1, 2005 and December 31, 2021. Patients' demographics, the cause for the injury, the diagnosis, the body region of the trauma and the treatment methods were collected. RESULTS: Two hundred and nine patients with injuries related to sauna bathing (83 female [39.7%] and 126 male [60.3%]) were identified. Fifty-one patients showed more than one injuries leading to a total of 274 diagnosis: contusions/distorsions (113; 41.2%), wounds (79; 28.8%), fractures (42; 15.3%), ligament injuries (17; 6.2%), concussions (15; 5.5%), burns (4; 1.5%) and brain bleeding (3; 1.1%). The most common cause for an injury was a slip/fall (157; 57.5%) followed by dizziness/syncope (82; 30.0%). Interestingly, head and face injuries were mostly caused by dizziness/syncope, whereas slip/fall was the leading cause for injuries of foot, hand, forearm and wrist. Nine patients(4.3%) needed surgical treatment mainly due to fractures. Eight patients got injured by wood splinters. One patient sustained grade IIB-III burns lying unconscious with an alcohol intoxication of 3.6‰ in the sauna. CONCLUSION: The main causes for injuries during sauna bathing were slip/falls and dizziness/syncopes. The latter one might be prevented by improved of the personal behaviour (e.g. drink enough water before and after each sauna bathing), whereas slip/falls might be prevented by the revision of safety regulations, particularly the obligation to wear slip resistant slippers. Thus, everyone himself as well as the operators can contribute to reduce injuries related sauna bathing.

Síncope por risa: descripción de un caso y revisión de la literatura / Laughter-induced syncope: description of a case and review of the literature

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Syncope and Raynaud's disease.

OBJECTIVE: To investigate an association between syncope and Raynaud's disease (RD), its clinical features, and the effect of treatment with nifedipine. DESIGN: One-year prospective study of new outpatients after 3 initial clinical observations. SETTING: Neurology clinics at Chelsea and Westminster, Royal Free, Barnet, and Edgware Hospitals. PATIENTS: Ten women and 1 man. The group had a mean (SD) age of 33 (17) years. Mean (SD) follow-up was 24 (36) months. INTERVENTION: Treatment with nifedipine. OUTCOME MEASURES: Observed vs expected frequency of syncope in RD, temporal relation between syncope and Raynaud's phenomenon, clinical features, and response to nifedipine treatment. RESULTS: Eight additional patients with syncope and RD were identified from 603 new patients (1.3%); we had expected only 1 patient to be identified with syncope and RD (P=.003). A chance association between RD and migraine with recurrent syncope was unlikely (P=.01). The prevalence of RD in patients with syncope with migraine was higher than expected (P=.03), but that of migraine in patients with RD was not (P=.2). All 11 patients had 5 or more syncopal episodes for a median of 2 years (range, 0.1-62 years). Three patients had previous diagnoses of nonepileptic attacks. Syncope was preceded by or contemporaneous with Raynaud's phenomenon in 10 patients (P=.02). Nine patients had migraine; headache was contemporaneous with syncope in 4 patients as expected by chance (P=1.0). In all patients, syncope was preceded by brainstem or vertebrobasilar symptoms, and it ceased after treatment with nifedipine. Raynaud's disease and migraine improved less. CONCLUSIONS: The association of syncope to RD was unrelated to chance or migraine. The temporal relation between syncope and Raynaud's phenomenon but not headache was statistically significant. Treatment with nifedipine stopped recurrent syncope in all patients. Syncope related to RD may result from brainstem ischemia. Unexplained recurrent syncope should prompt screening for RD.

Encefalopatía de Wernicke tras duodenopancreatectomía cefálica / Wernicke's encephalopathy after cephalic pancreaticoduodenectomy

La encefalopatía de Wernicke (EW) es un trastorno neurológico agudo resultado del déficit de tiamina. Presentamos la aparición de dicho cuadro en un enfermo joven que es sometido a una duodenopancreatectomía cefálica ante una úlcera duodenal sangrante refractaria a tratamiento endoscópico y quirúrgico previo, precisando de una nutrición parenteral total, sin suplementos de tiamina(AU)

Wernicke’s encephalopathy is an acute neurological disorder resulting from thiamine deficiency. We report a case in a young patient who underwent a cephalic duodenopancreatectomy with a bleeding duodenal ulcer refractory to endoscopic and surgical treatment, requiring total parenteral nutrition, without thiamine supplementation(AU)

Tratamento não-farmacológico da síncope neuromediada: [revisão] / Non-pharmacological treatment in neurally mediated syncope: [review]

A síncope neuromediada resulta de fenômeno transitório de hipofluxo cerebral. Normalmente está relacionada a uma condição benigna e passível de tratamento, farmacológico ou não. Diversas formas de tratamento não farmacológico têm sido propostas com a finalidade de obter maior controle sobre a frequência e a intensidade dos episódios de perda de consciência. O treinamento físico, o treinamento postural passivo e as contramanobras apresentam-se como alternativa terapêutica, visando promover maior adesão ao tratamento, por parte dos pacientes que relutam em fazer uso de medicação. O objetivo desta revisão é discorrer sobre as opções não-farmacológicas que envolvem exercício físico, para tratamento dessa entidade clínica, discutindo a fisiopatologia envolvida e as repercussões dos diversos tratamentos.

Abordaje diagnóstico y terapéutico del síncope en urgencias / Diagnostic and therapeutic approach of syncope in the emergency room

El síncope, definido como un cuadro de pérdida completa y transitoria de la conciencia con recuperación espontánea en un breve intervalo de tiempo debido a disminución del flujo sanguíneo cerebral, es un motivo frecuente de consulta en los servicios de urgencias. El síncope puede ser debido a múltiples causas con significado pronóstico muy variable. El papel del médico de urgencias consiste, en primer lugar, en diferenciar el síncope de otras causas de pérdida de conciencia, y luego intentar establecer la etiología más probable y perfilar la gravedad pronóstica, lo cual se puede conseguir mediante métodos sencillos y fácilmente disponibles. En este trabajo se describen someramente los diferentes mecanismos del síncope, los métodos para llegar a un diagnóstico etiológico, los criterios de ingreso hospitalario y el enfoque terapéutico (AU)

Syncope, defined as a transient lost of conciousness with spontaneous recovery in a short period of time due to an acute diminution of cerebral blood flow, is a frequent cause of admission in the emergency room. Syncope has multiple etiologies with very different pronostic implications. The role of the emergency room physician is, firstly, to differentiate syncope from nonsyncopal causes of lost of consciousness, and, secondly, to try to establish the most probable etiological diagnosis and its pronostic relevance by means of relatively simple and easily available tools. In this paper we describe the pathophysiology of syncope, its diagnostic approach and the criteria for hospital admission. Finally, some therapeutic recommendations are provided (AU)

Hiponatremia sintomática secundaria a tratamiento conjunto anticomicial y antidepresivo: ¿riesgo de muerte súbita en epilepsia? / Symptomatic secondary hyponatremia due to combined treatment anticonvulsant and antidepressant: risk of sudden death in epilepsy?

La notable incidencia de epilepsia en nuestro medio, con la presencia, cada vez más frecuente, de refractariedad terapéutica, obliga a la politerapia anticomicial. Si partimos del hecho, de que muchas de las epilepsias tienen una base secundaria, con trastornos comportamentales asociados, podemos comprender la cada vez más habitual asociación entre fármacos anticomiciales y antidepresivos. Presentamos el caso clínico de una mujer de 37 años, afecta de epilepsia farmacorresistente asociada a síndrome depresivo exógeno, en tratamiento conjunto con Carbamacepina, Lamotrigina y Venlafaxina que sufre cuadro sincopal secundario a hiponatremia sintomática, cuya patogénesis radica en un mecanismo de secreción inadecuada de hormona antidiurética (SIADH). La relevancia de este caso radica en el hecho de que la hiponatremia generada por esta combinación farmacológica, hasta ahora, no ha sido descrita en la literatura. Esta anomalía electrolítica puede derivar en efectos secundarios neurológicos y cardiovasculares, en ocasiones tan relevantes, como para producir el fenómeno conocido como muerte súbita en el paciente epiléptico (SUDEP). Recomendamos, por ello, un estricto control iónico en aquellos pacientes en tratamiento conjunto anticomicial y antidepresivo, a fin de evitar episodios paroxísticos vasculares y minimizar el riesgo de SUDEP

The notable incidence of epilepsy in the general population, with the presence, more and more frequent of refractory therapeutic, it forces the anticonvulsant polytherapy. If we depart from the fact, that many of the epilepsies have a secondary cause, with affective disorders associate, we can understand the most habitual association of anticonvulsant and antidepressant. We present the clinical case of a 37-year-old-woman with refractory therapeutic epilepsy associated to exogenous depressive syndrome, in combined treatment with Carbamazepine, Lamotrigine and Venlafaxine, that suffers a episode of syncope secondary to symptomatic hyponatraemia, generated by syndrome of inappropriate secretion of antidiuretic hormone (SIADH). The relevance of this case resides in the fact that the hyponatraemia generated by this pharmacological combination, up to now, it has not been described in the literature. This electrolytic anomaly can derive in secondary neurological and cardiovascular effects, in so outstanding occasions, as to produce the phenomenon denominated sudden death in the epileptic patient (SUDEP). We recommended a strict ionic control in those patients that require anti-epileptic and anti-depressant treatment combined, in order to avoiding paroxistic vascular episodes and to minimize the risk of SUDEP

Transesophageal electrophysiologic study in children and young patients.

Transesophageal electrophysiologic study (TEEPS) is a semi-invasive method of atrial stimulation and recording. The aim of the study was to report our experience with TEEPS in children and young adults. A total of 153 TEEPS were performed in 147 consecutive patients aged between 26 days to 26 years (mean 9.8 years) with the following indications: evaluation of symptoms that may be signs of any arrhythmias in 89 procedures (Group A), risk assessment of Wolff-Parkinson-White syndrome (WPW) in 17 procedures (Group B), determination of the mechanism of previously detected or ongoing tachycardia on ECG or Holter monitoring in 22 procedures (Group C), assessment of antiarrhythmic therapy effectiveness in 17 procedures (Group D), and follow-up of radiofrequency ablation procedure (RFA) in 8 procedures (Group E). A similar pacing protocol was performed for induction of tachycardia in each patient. Tachycardia was induced in a total of 72 procedures (72/153, 47%): 32/89 (36%) in Group A, 13/17 (76.5%) in Group B, 12/22 (54.5%) in Group C, 12/17 (70.6%) in Group D and 3/8 (37.5%) in Group E. In Group A, the ventriculoatrial (VA) interval of inducible tachycardia was found to be shorter than 70 msec in 16/32 (50%) and longer than 70 msec in 12/32 (37.5%) patients and these patients were diagnosed as having atrioventricular nodal reentrant tachycardia (AVNRT) and atrioventricular reentrant tachycardia (AVRT), respectively. In this group, 1 atrial tachycardia, 2 junctional ectopic tachycardia, 1 sinus node reentrant tachycardia and 1 permanent junctional reciprocating tachycardia (PJRT) were also diagnosed. In conclusion, transesophageal atrial stimulation is a valuable tool in the initial evaluation of patients with symptoms possibly related with arrhythmia or in the management of patients who have any arrhythmia.

Usefulness of prolonged QRS duration to identify high-risk ischemic cardiomyopathy patients with syncope and inducible ventricular tachycardia.

We evaluated 61 consecutive patients who had coronary artery disease, decreased left ventricular function, and syncope and underwent implantation of a cardioverter-defibrillator because sustained ventricular tachycardia was inducible at electrophysiologic testing. During a follow-up of 3.0 +/- 1.8 years, 23 patients (38%) developed ventricular tachycardia. Prolonged QRS duration (>/=120 ms) was the only significant predictor of arrhythmia. The 1- and 2-year rates without ventricular arrhythmia were 82% and 77%, respectively, in patients whose QRS duration was <120 ms. In contrast, 1- and 2-year rates without ventricular arrhythmia were only 64% and 51%, respectively, in patients whose QRS duration was >/=120 ms (risk ratio 3.7, 95% confidence interval 1.4 to 9.8, p = 0.0092).

Spontaneous ventricular tachycardia and fibrillation in a patient with a positive microvolt T wave alternans test and negative electrophysiological study.

This report describes a patient with a previous myocardial infarction who presented with syncope. The patient had a positive microvolt T wave alternans test, a negative electrophysiological study, and a normal heart rate variability. In hospital, the patient had episodes of ventricular tachycardia and fibrillation. An implantable cardioverter defibrillator was implanted and during the following week it discharged appropriately.

Inducible ventricular flutter and fibrillation predict for arrhythmia occurrence in coronary artery disease patients presenting with syncope of unknown origin.

INTRODUCTION: Ventricular fibrillation and ventricular flutter (cycle length < or = 230 msec) induced at electrophysiologic studies are thought to be nonspecific findings in patients presenting with syncope of unknown origin. However, there are limited data on the prognosis of these patients in long-term follow-up. METHODS AND RESULTS: We followed 274 consecutive patients with coronary artery disease presenting with syncope or presyncope who underwent electrophysiologic studies from January 1992 to June 1999 and assessed the risk of subsequent arrhythmias stratified by the electrophysiologic result at the time of their presentation with syncope. Ventricular fibrillation was induced in 23 patients (8%); ventricular flutter in 24 (9%), sustained ventricular tachycardia in 41 (15%); and nonsustained ventricular tachycardia 42 (15%). In 37 +/- 25 months of follow-up, there have been ventricular arrhythmias in 34 patients, including 3 (13%) of 23 who had induced ventricular fibrillation, and 7 (30%) of 24 with induced ventricular flutter, compared to 13 (32%) of 41 with sustained ventricular tachycardia, 7 (17%) of 42 with nonsustained ventricular tachycardia, and only 4 (3%) of 144 noninducible patients (P < 0.001 for induced ventricular fibrillation and ventricular flutter vs noninducible patients). The inducibility of ventricular fibrillation and ventricular flutter were independent risk factors for arrhythmia occurrence in follow-up. CONCLUSION: Ventricular fibrillation and ventricular flutter induced at electrophysiologic studies have prognostic significance for arrhythmia occurrence in patients presenting with syncope. These induced arrhythmias may not be as nonspecific as previously thought and treatment should be considered for these patients.

Convulsive syncope associated with acupuncture.

Syncope is a rare but known reaction to acupuncture; however, convulsive syncope has never been previously documented as a reaction to acupuncture. This case report describes an episode of convulsive syncope, characterized by irregular clonic-tonic movements while the patient was unconscious. The episode occurred immediately after the insertion of acupuncture needles into the bilateral ST-36 acupuncture point. Here we discuss the presentation, possible causes, and prevention of convulsive syncope.

Significance of inducible ventricular fibrillation in patients with coronary artery disease and unexplained syncope.

OBJECTIVES: This study was designed to determine the incidence and prognostic significance of inducible ventricular fibrillation (VF) in patients with coronary artery disease (CAD) and unexplained syncope. BACKGROUND: Current American College of Cardiology/American Heart Association practice guidelines recommend implantation of internal cardioverter-defibrillators (ICDs) in patients with unexplained syncope in whom either ventricular tachycardia (VT) or VF is inducible during electrophysiologic (EP) testing. Although the prognostic significance of inducible monomorphic VT is known, the significance of inducible VF remains undefined. METHODS: We evaluated 118 consecutive patients with CAD and unexplained syncope who underwent EP testing. Sustained monomorphic VT was inducible in 53 (45%) patients; in 20 (17%) patients, VF was the only inducible arrhythmia; and no sustained ventricular arrhythmia was inducible in the remaining 45 (38%) patients. The latter two groups of 65 (55%) patients make up the study population. RESULTS: There were 16 deaths among the study population during a follow-up period of 25.3 +/- 19.6 months. The overall one- and two-year survival in these patients was 89% and 81%, respectively. No significant difference in survival was observed between patients with and without inducible VF (80% power to detect a fourfold survival difference). CONCLUSIONS: In 17% of patients with CAD and unexplained syncope, VF is the only inducible ventricular arrhythmia. Within the limits of this pilot study, long-term follow-up of patients with and without inducible VF demonstrates no difference in survival between the two groups. Therefore, the practice of ICD implantation in patients with CAD, unexplained syncope and inducible VF, especially with triple ventricular extrastimuli, may merit reconsideration.

Syncope in patients with atrial flutter during treatment with class Ic antiarrhythmic drugs.

We describe 2 atrial flutter (AFL) patients with syncope during treatment with class Ic antiarrhythmic drugs. During the syncope, 1:1 atrioventricular (AV) conduction during AFL preceded a wide QRS tachycardia. The class Ic drugs, flecainide and pilsicainide, slowed the atrial rate, resulting in AFL with 1:1 AV conduction, and the width of the QRS complexes became wider during the tachycardia. Syncope was abolished after successful radiofrequency catheter ablation of the AFL. These potential proarrhythmic effects of the class Ic drugs should be taken into account in AFL patients, and concomitant use of beta-blocking agents would be critical to prevent proarrhythmias.