Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria.

Kowaltowski, A J; Seetharaman, S; Paucek, P; Garlid, K D

Kowaltowski, A J; Seetharaman, S; Paucek, P; Garlid, K D.

Affiliation

Kowaltowski AJ; Department of Biochemistry and Molecular Biology, Oregon Graduate Institute of Science and Technology, Beaverton, Oregon 97006-8921, USA.

Am J Physiol Heart Circ Physiol ; 280(2): H649-57, 2001 Feb.

Article in En | MEDLINE | ID: mdl-11158963

Subject(s)

Adenosine Triphosphate/metabolism; Energy Metabolism/physiology; Mitochondria/metabolism; Myocardial Reperfusion Injury/metabolism; Myocardium/metabolism; Potassium Channels/metabolism; Animals; Anions/metabolism; Cell Respiration/drug effects; Cell Respiration/physiology; Diazoxide/toxicity; Energy Metabolism/drug effects; Ion Channel Gating/drug effects; Ion Channel Gating/physiology; Membrane Potentials/physiology; Mitochondria/drug effects; Mitochondrial Swelling/drug effects; Mitochondrial Swelling/physiology; Pinacidil/toxicity; Potassium/metabolism; Rats; Succinic Acid/metabolism; Uncoupling Agents/toxicity; Vasodilator Agents/toxicity

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Database: MEDLINE Therapeutic Methods and Therapies TCIM: Terapias_energeticas / Bioenergetica Main subject: Myocardial Reperfusion Injury / Potassium Channels / Adenosine Triphosphate / Energy Metabolism / Mitochondria / Myocardium Type of study: Diagnostic_studies Language: En Journal: Am J Physiol Heart Circ Physiol Year: 2001 Type: Article Affiliation country: United States

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