Cutting edge: Stat6-dependent substrate depletion regulates nitric oxide production.

Rutschman, R; Lang, R; Hesse, M; Ihle, J N; Wynn, T A; Murray, P J

Rutschman, R; Lang, R; Hesse, M; Ihle, J N; Wynn, T A; Murray, P J.

Affiliation

Rutschman R; Department of Infectious Diseases, Howard Hughes Medical Institute, St. Jude Children's Research Hospital, Memphis, TN 38105, USA.

J Immunol ; 166(4): 2173-7, 2001 Feb 15.

Article in En | MEDLINE | ID: mdl-11160269

Subject(s)

Nitric Oxide/biosynthesis; Trans-Activators/physiology; Animals; Arginase/biosynthesis; Arginine/deficiency; Cells, Cultured; Down-Regulation/immunology; Interleukin-13/physiology; Interleukin-4/physiology; Macrophage Activation; Macrophages, Peritoneal/enzymology; Macrophages, Peritoneal/immunology; Macrophages, Peritoneal/metabolism; Macrophages, Peritoneal/parasitology; Mice; Mice, Inbred C57BL; Mice, Knockout; Nitric Oxide/antagonists & inhibitors; Nitric Oxide Synthase/metabolism; Nitric Oxide Synthase Type II; STAT6 Transcription Factor; Substrate Specificity/immunology; Toxoplasmosis, Animal/enzymology; Toxoplasmosis, Animal/immunology; Toxoplasmosis, Animal/metabolism; Trans-Activators/deficiency; Trans-Activators/genetics

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Database: MEDLINE Main subject: Trans-Activators / Nitric Oxide Language: En Journal: J Immunol Year: 2001 Type: Article Affiliation country: United States

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Database: MEDLINE Main subject: Trans-Activators / Nitric Oxide Language: En Journal: J Immunol Year: 2001 Type: Article Affiliation country: United States