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Enhanced activation of STAT pathways and overexpression of survivin confer resistance to FLT3 inhibitors and could be therapeutic targets in AML.
Zhou, Jianbiao; Bi, Chonglei; Janakakumara, Jasinghe V; Liu, Shaw-Cheng; Chng, Wee-Joo; Tay, Kian-Ghee; Poon, Lai-Fong; Xie, Zhigang; Palaniyandi, Senthilnathan; Yu, Hanry; Glaser, Keith B; Albert, Daniel H; Davidsen, Steven K; Chen, Chien-Shing.
Affiliation
  • Zhou J; Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.
Blood ; 113(17): 4052-62, 2009 Apr 23.
Article in En | MEDLINE | ID: mdl-19144991
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Full text: 1 Database: MEDLINE Therapeutic Methods and Therapies TCIM: Terapias_biologicas / Aromoterapia Main subject: Leukemia, Myeloid, Acute / Signal Transduction / Gene Expression Regulation, Neoplastic / Fms-Like Tyrosine Kinase 3 / STAT3 Transcription Factor / Microtubule-Associated Proteins Type of study: Prognostic_studies Language: En Journal: Blood Year: 2009 Type: Article Affiliation country: Singapore
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Full text: 1 Database: MEDLINE Therapeutic Methods and Therapies TCIM: Terapias_biologicas / Aromoterapia Main subject: Leukemia, Myeloid, Acute / Signal Transduction / Gene Expression Regulation, Neoplastic / Fms-Like Tyrosine Kinase 3 / STAT3 Transcription Factor / Microtubule-Associated Proteins Type of study: Prognostic_studies Language: En Journal: Blood Year: 2009 Type: Article Affiliation country: Singapore