Glucose stimulation of hypothalamic MCH neurons involves K(ATP) channels, is modulated by UCP2, and regulates peripheral glucose homeostasis.

Kong, Dong; Vong, Linh; Parton, Laura E; Ye, Chianping; Tong, Qingchun; Hu, Xiaoxia; Choi, Brian; Brüning, Jens C; Lowell, Bradford B

Kong, Dong; Vong, Linh; Parton, Laura E; Ye, Chianping; Tong, Qingchun; Hu, Xiaoxia; Choi, Brian; Brüning, Jens C; Lowell, Bradford B.

Affiliation

Kong D; Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA.

Cell Metab ; 12(5): 545-52, 2010 Nov 03.

Article in En | MEDLINE | ID: mdl-21035764

Subject(s)

Glucose/metabolism; Hypothalamic Hormones/metabolism; Hypothalamus/cytology; Ion Channels/metabolism; KATP Channels/metabolism; Melanins/metabolism; Mitochondrial Proteins/metabolism; Neurons/metabolism; Pituitary Hormones/metabolism; Animals; Cells, Cultured; Gene Expression; Ion Channels/genetics; KATP Channels/genetics; Mice; Mice, Transgenic; Mitochondrial Proteins/genetics; Mutation; Uncoupling Protein 2

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Full text: 1 Database: MEDLINE Main subject: Pituitary Hormones / Mitochondrial Proteins / KATP Channels / Glucose / Hypothalamic Hormones / Hypothalamus / Ion Channels / Melanins / Neurons Language: En Journal: Cell Metab Year: 2010 Type: Article Affiliation country: United States

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