Angular Momentum Transport by Keplerian Turbulence in Liquid Metals.

We report a laboratory study of the transport of angular momentum by a turbulent flow of an electrically conducting fluid confined in a thin disk. When the electromagnetic force applied to the liquid metal is large enough, the corresponding volume injection of angular momentum produces a turbulent flow characterized by a time-averaged Keplerian rotation rate Ω[over ¯]∼r^{-3/2}. Two contributions to the local angular momentum transport are identified: one from the poloidal recirculation induced by the presence of boundaries and the other from turbulent fluctuations in the bulk. The latter produces efficient angular momentum transport independent of the molecular viscosity of the fluid and leads to Kraichnan's prediction Nu_{Ω}∝sqrt[Ta]. In this so-called ultimate regime, the experiment, therefore, provides a configuration analogous to accretion disks, allowing the prediction of accretion rates induced by Keplerian turbulence.

1/f noise and long-term memory of coherent structures in a turbulent shear flow.

A shear flow of liquid metal (Galinstan) is driven in an annular channel by counter-rotating traveling magnetic fields imposed at the end caps. When the traveling velocities are large, the flow is turbulent and its azimuthal component displays random reversals. Power spectra of the velocity field exhibit a 1/f^{α} power law on several decades and are related to power-law probability distributions P(τ)∼τ^{-ß} of the waiting times between successive reversals. This 1/f type spectrum is observed only when the Reynolds number is large enough. In addition, the exponents α and ß are controlled by the symmetry of the system; a continuous transition between two different types of Flicker noise is observed as the equatorial symmetry of the flow is broken, in agreement with theoretical predictions.

Fluctuations of Electrical Conductivity: A New Source for Astrophysical Magnetic Fields.

We consider the generation of a magnetic field by the flow of a fluid for which the electrical conductivity is nonuniform. A new amplification mechanism is found which leads to dynamo action for flows much simpler than those considered so far. In particular, the fluctuations of the electrical conductivity provide a way to bypass antidynamo theorems. For astrophysical objects, we show through three-dimensional global numerical simulations that the temperature-driven fluctuations of the electrical conductivity can amplify an otherwise decaying large scale equatorial dipolar field. This effect could play a role for the generation of the unusually tilted magnetic field of the iced giants Neptune and Uranus.

Activation of human monocyte-derived macrophages by immune complexes containing low-density lipoprotein.

Human monocyte-derived macrophages are transformed into foam cells upon incubation with immune complexes containing low-density lipoprotein (LDL-IC), which are internalized predominantly through Fc gamma receptor-mediated phagocytosis. We investigated whether the FcR gamma-mediated ingestion of LDL-IC is associated with functional and metabolic activation of the ingesting cells. As end points we used the assay of released interleukin-1 beta (IL-1 beta) and tumor necrosis factor alpha (TNF alpha) and the reduction of nitroblue tetrazolium, which measures the respiratory burst. LDL-IC, added to the macrophages in concentrations known to induce intracellular accumulation of cholesterol esters and foam cell transformation, stimulated both the cytokine release and the respiratory burst more efficiently than control immune complexes. Time course studies of cytokine release and mRNA expression suggest that the synthesis and release of these two cytokines is under independent control. TNF alpha was released almost immediately after addition of LDL-IC to the macrophages, coinciding with increased early expression of TNF alpha mRNA, detectable 30 min after stimulation. In contrast, IL-1 beta was only increased in stimulated cell supernatants after 8 hr, and the onset of expression of IL-1 beta mRNA was also delayed in comparison to that of TNF alpha mRNA. We noted wide variations in the amounts of TNF alpha released by monocyte-derived macrophages from different donors. We also found that those macrophages which released higher levels of TNF alpha also took up higher amounts of 125I-labeled LDL, suggesting that the expression of LDL receptors by LDL-IC-stimulated macrophages is somehow linked to the degree of activation of these cells. Experiments using the measurement of the oxidative burst as end point corroborated that LDL-IC cause a general activation of macrophage functions. In conclusion, human macrophages are efficiently activated by LDL-IC, as reflected by the release of IL-1 beta and TNF alpha and by the release of oxygen active radicals. Thus, the presentation of LDL-IC to human macrophages induces a variety of metabolic and functional changes which are likely to contribute, directly or indirectly, to endothelial damage and progression of the atheromatous lesion.