Asunto(s)
Acetaminofén/efectos adversos , Analgésicos no Narcóticos/efectos adversos , Erupciones por Medicamentos/etiología , Hipersensibilidad a las Drogas/inmunología , Fenilefrina/efectos adversos , Simpatomiméticos/efectos adversos , Acetaminofén/administración & dosificación , Acetaminofén/química , Adulto , Analgésicos no Narcóticos/administración & dosificación , Pruebas de Provocación Bronquial , Resfriado Común/tratamiento farmacológico , Resfriado Común/inmunología , Resfriado Común/fisiopatología , Reacciones Cruzadas , Hipersensibilidad a las Drogas/fisiopatología , Femenino , Cefalea/prevención & control , Humanos , Pruebas del Parche , Fenilefrina/administración & dosificación , Fenilefrina/química , Simpatomiméticos/administración & dosificaciónRESUMEN
OBJECTIVES: To assess changes in the serum cholesterol (CT) during acute coronary events (infarction -IAM- and unstable angina) in clinical practice, to raise to begin pharmacologic lipid therapy knowing only a lipid profile. METHODS: Lipid clinic database was revised. SELECTION CRITERIA: patients who were sent to the clinic 1 to 3 months after the acute coronary event and who were not begun pharmacologic lipid therapy. RESULTS: 56 patients, 69% with IAM. Admission CT mean was lower. Among 17 who had CT lower than 200 mg/dl during acute phase, 16 had CT higher than 200 in ambulatory lipid profile. The bigger acute phase drop took place between those who had CT higher during ambulatory consultation. CONCLUSIONS: Theses results confirm the CT decrease during IAM and reports this reduction happens during unstable angina too. That permits to suggest mostly to begin the lipid lowering drug therapy earlier, even although there was only a lipid value known.
Asunto(s)
Enfermedad de la Arteria Coronaria/sangre , Enfermedad de la Arteria Coronaria/prevención & control , Hipolipemiantes/uso terapéutico , Lípidos/sangre , Adulto , Anciano , Angina Inestable/sangre , Femenino , Humanos , Masculino , Persona de Mediana Edad , Infarto del Miocardio/sangreAsunto(s)
Mioclonía/etiología , Neurosífilis/diagnóstico , Demencia/etiología , Humanos , Masculino , Persona de Mediana EdadRESUMEN
An increasing QT interval can precipitate life-threatening tachyarrhythmias such as ventricular fibrillation. Tuberculous myocarditis is a very unusual diagnosis commonly made at autopsy. Mycobacterium tuberculosis can invade the cardiac conduction system and produce potentially dangerous arrhythmias. This case presents an HIV-infected man with tuberculous infection and long QT syndrome. We comment on the pathology, clinical features and outcome of this rare form of tuberculous infection.
RESUMEN
OBJECTIVE: To know the frequency of resistances of Mycobacterium tuberculosis and the associated factors. PATIENTS AND METHODS: Prospective study of the sensitivity of Mycobacterium tuberculosis by means of the method of the proportions of Canetti in the Hospital Arquitecto Marcide-Profesor Novoa Santos (Ferrol, Spain) among 1991 and 1998. A descriptive and multiple regression analyses were performed. RESULTS: Were studied 355 strains. Primary resistance: Isoniazid 1.1%, Streptomycin 1.1%. Secondary resistance: Isoniazid 11.6%, in the 5.2% existed multidrug-resistance. The risk factors for drug-resistant tuberculosis were previous treatment (odds ratio [OR] = 10.9; 95% CI, 2.9-39.4) and age higher than 40 years (OR = 3.9; 95% CI, 1.1-14.5). CONCLUSIONS: A low frequency of resistance was observed. The factors associated with the resistances were previous treatment and age.