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Transient inhibition of the ERK pathway prevents cerebellar developmental defects and improves long-term motor functions in murine models of neurofibromatosis type 1.
Kim, Edward; Wang, Yuan; Kim, Sun-Jung; Bornhorst, Miriam; Jecrois, Emmanuelle S; Anthony, Todd E; Wang, Chenran; Li, Yi E; Guan, Jun-Lin; Murphy, Geoffrey G; Zhu, Yuan.
Afiliación
  • Kim E; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Wang Y; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Kim SJ; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Bornhorst M; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Jecrois ES; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Anthony TE; Laboratory of Molecular Biology, Rockefeller University, New York, United States.
  • Wang C; Department of Cancer Biology, University of Cincinnati College of Medicine, Cincinnati, United States.
  • Li YE; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Guan JL; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
  • Murphy GG; Molecular and Behavioral Neuroscience Institute, University of Michigan Medical School, Ann Arbor, United States.
  • Zhu Y; Division of Molecular Medicine and Genetics, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, United States.
Elife ; 32014 Dec 23.
Article en En | MEDLINE | ID: mdl-25535838
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Neurofibromatosis / Quinasas MAP Reguladas por Señal Extracelular Tipo de estudio: Prognostic_studies Idioma: En Revista: Elife Año: 2014 Tipo del documento: Article
Texto completo
Imprimir
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PubMed Links
Buscar en Google

Texto completo: 1 Base de datos: MEDLINE Asunto principal: Neurofibromatosis / Quinasas MAP Reguladas por Señal Extracelular Tipo de estudio: Prognostic_studies Idioma: En Revista: Elife Año: 2014 Tipo del documento: Article