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Mitochondrial impairments contribute to Spinocerebellar ataxia type 1 progression and can be ameliorated by the mitochondria-targeted antioxidant MitoQ.
Stucki, David M; Ruegsegger, Céline; Steiner, Silvio; Radecke, Julika; Murphy, Michael P; Zuber, Benoît; Saxena, Smita.
Afiliación
  • Stucki DM; Institute of Cell Biology, University of Bern, Bern, Switzerland; Graduate School for Cellular and Biomedical Sciences, University of Bern, Bern, Switzerland.
  • Ruegsegger C; Institute of Cell Biology, University of Bern, Bern, Switzerland; Graduate School for Cellular and Biomedical Sciences, University of Bern, Bern, Switzerland.
  • Steiner S; Institute of Cell Biology, University of Bern, Bern, Switzerland.
  • Radecke J; Institute of Anatomy, University of Bern, Bern, Switzerland; Graduate School for Cellular and Biomedical Sciences, University of Bern, Bern, Switzerland.
  • Murphy MP; Medical Research Council, Mitochondrial Biology Unit, Cambridge, United Kingdom.
  • Zuber B; Institute of Anatomy, University of Bern, Bern, Switzerland.
  • Saxena S; Institute of Cell Biology, University of Bern, Bern, Switzerland. Electronic address: smita.saxena@izb.unibe.ch.
Free Radic Biol Med ; 97: 427-440, 2016 08.
Article en En | MEDLINE | ID: mdl-27394174

Texto completo: 1 Base de datos: MEDLINE Asunto principal: Compuestos Organofosforados / Ubiquinona / Ataxias Espinocerebelosas / Antioxidantes Idioma: En Revista: Free Radic Biol Med Asunto de la revista: BIOQUIMICA / MEDICINA Año: 2016 Tipo del documento: Article

Texto completo: 1 Base de datos: MEDLINE Asunto principal: Compuestos Organofosforados / Ubiquinona / Ataxias Espinocerebelosas / Antioxidantes Idioma: En Revista: Free Radic Biol Med Asunto de la revista: BIOQUIMICA / MEDICINA Año: 2016 Tipo del documento: Article