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Gene therapy for Alzheimer's disease targeting CD33 reduces amyloid beta accumulation and neuroinflammation.
Griciuc, Ana; Federico, Anthony N; Natasan, Jeyashree; Forte, Angela M; McGinty, Danielle; Nguyen, Huong; Volak, Adrienn; LeRoy, Stanley; Gandhi, Sheetal; Lerner, Eli P; Hudry, Eloise; Tanzi, Rudolph E; Maguire, Casey A.
Afiliación
  • Griciuc A; Genetics and Aging Research Unit, McCance Center for Brain Health, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02129, USA.
  • Federico AN; Genetics and Aging Research Unit, McCance Center for Brain Health, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02129, USA.
  • Natasan J; Molecular Neurogenetics Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02114, USA.
  • Forte AM; Genetics and Aging Research Unit, McCance Center for Brain Health, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02129, USA.
  • McGinty D; Genetics and Aging Research Unit, McCance Center for Brain Health, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02129, USA.
  • Nguyen H; Alzheimer's Disease Research Laboratory, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02129, USA.
  • Volak A; Molecular Neurogenetics Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02114, USA.
  • LeRoy S; Molecular Neurogenetics Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02114, USA.
  • Gandhi S; Alzheimer's Disease Research Laboratory, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02129, USA.
  • Lerner EP; Alzheimer's Disease Research Laboratory, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02129, USA.
  • Hudry E; Alzheimer's Disease Research Laboratory, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02129, USA.
  • Tanzi RE; Genetics and Aging Research Unit, McCance Center for Brain Health, MassGeneral Institute for Neurodegenerative Disease, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA 02129, USA.
  • Maguire CA; Molecular Neurogenetics Unit, Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Charlestown, MA, 02114, USA.
Hum Mol Genet ; 29(17): 2920-2935, 2020 10 10.
Article en En | MEDLINE | ID: mdl-32803224
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Terapia Genética / Presenilina-1 / Enfermedad de Alzheimer / Lectina 3 Similar a Ig de Unión al Ácido Siálico Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2020 Tipo del documento: Article
Texto completo
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Terapia Genética / Presenilina-1 / Enfermedad de Alzheimer / Lectina 3 Similar a Ig de Unión al Ácido Siálico Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2020 Tipo del documento: Article