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TFEB/Mitf links impaired nuclear import to autophagolysosomal dysfunction in C9-ALS.
Cunningham, Kathleen M; Maulding, Kirstin; Ruan, Kai; Senturk, Mumine; Grima, Jonathan C; Sung, Hyun; Zuo, Zhongyuan; Song, Helen; Gao, Junli; Dubey, Sandeep; Rothstein, Jeffrey D; Zhang, Ke; Bellen, Hugo J; Lloyd, Thomas E.
Afiliación
  • Cunningham KM; Cellular and Molecular Medicine Program, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Maulding K; Cellular and Molecular Medicine Program, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Ruan K; Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Senturk M; Program in Developmental Biology, Baylor College of Medicine (BCM), Houston, United States.
  • Grima JC; Brain Science Institute, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Sung H; Solomon H. Snyder Department of Neuroscience, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Zuo Z; Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Song H; Department of Molecular and Human Genetics, BCM, Houston, United States.
  • Gao J; Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Dubey S; Department of Neuroscience, Mayo Clinic, Jacksonville, United States.
  • Rothstein JD; Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Zhang K; Cellular and Molecular Medicine Program, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Bellen HJ; Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, United States.
  • Lloyd TE; Brain Science Institute, School of Medicine, Johns Hopkins University, Baltimore, United States.
Elife ; 92020 12 10.
Article en En | MEDLINE | ID: mdl-33300868
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Autofagia / Transporte Activo de Núcleo Celular / Factores de Transcripción Básicos con Cremalleras de Leucinas y Motivos Hélice-Asa-Hélice / Factor de Transcripción Asociado a Microftalmía Tipo de estudio: Prognostic_studies Idioma: En Revista: Elife Año: 2020 Tipo del documento: Article
Texto completo
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Autofagia / Transporte Activo de Núcleo Celular / Factores de Transcripción Básicos con Cremalleras de Leucinas y Motivos Hélice-Asa-Hélice / Factor de Transcripción Asociado a Microftalmía Tipo de estudio: Prognostic_studies Idioma: En Revista: Elife Año: 2020 Tipo del documento: Article