NPD1 Enhances Autophagy and Reduces Hyperphosphorylated Tau and Amyloid-ß42 by Inhibiting GSK3ß Activation in N2a/APP695swe Cells.

Dai, Songyang; Zhou, Fanlin; Sun, Jieyun; Li, Yu

Dai, Songyang; Zhou, Fanlin; Sun, Jieyun; Li, Yu.

Afiliación

Dai S; Institute of Neuroscience, School of Basic Medicine, Chongqing Medical University, Chongqing, China.
Zhou F; Department of Pathology, Chongqing University Cancer Hospital & Chongqing Cancer Institute & Chongqing Cancer Hospital, Chongqing, China.
Sun J; Institute of Neuroscience, School of Basic Medicine, Chongqing Medical University, Chongqing, China.
Li Y; Institute of Neuroscience, School of Basic Medicine, Chongqing Medical University, Chongqing, China.

J Alzheimers Dis ; 84(2): 869-881, 2021.

Article en En | MEDLINE | ID: mdl-34602482

Asunto(s)

Enfermedad de Alzheimer/metabolismo; Autofagia; Ácidos Docosahexaenoicos/metabolismo; Glucógeno Sintasa Quinasa 3 beta/metabolismo; Proteínas tau; Péptidos beta-Amiloides; Animales; Apoptosis; Técnicas de Cultivo de Célula; Humanos; Ratones; Fármacos Neuroprotectores; Fragmentos de Péptidos; Fosforilación

Palabras clave

Alzheimer's disease; Neuroprotectin D1; autophagy; glycogen synthase kinase 3

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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Autofagia / Ácidos Docosahexaenoicos / Proteínas tau / Enfermedad de Alzheimer / Glucógeno Sintasa Quinasa 3 beta Idioma: En Revista: J Alzheimers Dis Asunto de la revista: GERIATRIA / NEUROLOGIA Año: 2021 Tipo del documento: Article

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