Macrophages in periodontitis: A dynamic shift between tissue destruction and repair.

Yin, Linying; Li, Xinzhu; Hou, Jin.

Afiliación

Yin L; Department of Stomatology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Li X; Department of Stomatology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Hou J; Department of Stomatology, Nanfang Hospital, Southern Medical University, Guangzhou, China.

Jpn Dent Sci Rev ; 58: 336-347, 2022 Nov.

Article en En | MEDLINE | ID: mdl-36340583

Palabras clave

AIM2, absent in melanoma-2; CCL2, chemokine (C-C motif) ligand 2; CCR2, chemokine (C-C motif) receptor 2; DAMP, damage-associated molecular pattern; Dysbiosis; IFN, interferon; IL, interleukin; IRF, interferon regulatory factor; Inflammation; LPS, lipopolysaccharide; MLKL, mixed lineage kinase-like; MMP, matrix metalloproteinase; Macrophages; MyD88, myeloid differentiation factor 88; NLR, nucleotide oligomerization domain-like receptor; Oral microbiome; PAMP, pathogen-associated molecular pattern; PRR, pattern recognition receptor; Periodontitis; RANKL, receptor activator of nuclear factor kappa-B ligand; STAT, signal transducer and activator of transcription; TGF, transforming growth factor; TLR, toll-like receptor; TNF, tumor necrosis factor; TRIF, Toll/IL-1R domain-containing adapter-inducing IFN-ß