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Blocking IL-6 signaling improves glucose tolerance via SLC39A5-mediated suppression of glucagon secretion.
Chen, Wenli; Cui, Weiyi; Wu, Jianhong; Zheng, Wen; Sun, Xueting; Zhang, Jie; Shang, Haibao; Yuan, Ye; Li, Xue; Wang, Jue; Hu, Xinli; Chen, Liangyi; Zeng, Fanxin; Xiao, Rui-Ping; Zhang, Xiuqin.
Afiliación
  • Chen W; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Cui W; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Wu J; Department of Rheumatology and Immunology, Dazhou Central Hospital, Dazhou, China.
  • Zheng W; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Sun X; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Zhang J; Department of Clinical Research Center, Dazhou Central Hospital, Dazhou 635000, China.
  • Shang H; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Yuan Y; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Li X; Department of Clinical Research Center, Dazhou Central Hospital, Dazhou 635000, China.
  • Wang J; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Hu X; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China.
  • Chen L; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China; National Biomedical Imaging Center, School of Future Technology, Peking University, Beijing
  • Zeng F; Department of Clinical Research Center, Dazhou Central Hospital, Dazhou 635000, China. Electronic address: zengfx@pku.edu.cn.
  • Xiao RP; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China; State Key Laboratory of Biomembrane and Membrane Biotechnology, Peking-Tsinghua Center for
  • Zhang X; Institute of Molecular Medicine, College of Future Technology, Peking University, Beijing 100871, China; Beijing Key Laboratory of Cardiometabolic Molecular Medicine, Peking University, Beijing 100871, China; National Biomedical Imaging Center, School of Future Technology, Peking University, Beijing
Metabolism ; 146: 155641, 2023 09.
Article en En | MEDLINE | ID: mdl-37380017
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Resistencia a la Insulina / Proteínas de Transporte de Catión / Diabetes Mellitus Tipo 2 / Células Secretoras de Glucagón Idioma: En Revista: Metabolism Año: 2023 Tipo del documento: Article
Texto completo
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Resistencia a la Insulina / Proteínas de Transporte de Catión / Diabetes Mellitus Tipo 2 / Células Secretoras de Glucagón Idioma: En Revista: Metabolism Año: 2023 Tipo del documento: Article