The E3 ubiquitin ligase TRAF6 controls CTLA-4 turnover and promotes T-cell-mediated antitumor immunity.

Jie, Xiao-Liang; Guo, Hua; Zhou, Guang-Biao

Jie, Xiao-Liang; Guo, Hua; Zhou, Guang-Biao.

Afiliación

Jie XL; State Key Laboratory of Molecular Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, 100021, Beijing, China.
Guo H; State Key Laboratory of Molecular Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, 100021, Beijing, China.
Zhou GB; State Key Laboratory of Molecular Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, 100021, Beijing, China. gbzhou@cicams.ac.cn.

Cell Mol Immunol ; 21(1): 97-99, 2024 01.

Article en En | MEDLINE | ID: mdl-38082145

Asunto(s)

Factor 6 Asociado a Receptor de TNF; Ubiquitina-Proteína Ligasas; Ubiquitina-Proteína Ligasas/metabolismo; Factor 6 Asociado a Receptor de TNF/metabolismo; Antígeno CTLA-4; Linfocitos T/metabolismo; Ubiquitinación

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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Ubiquitina-Proteína Ligasas / Factor 6 Asociado a Receptor de TNF Idioma: En Revista: Cell Mol Immunol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2024 Tipo del documento: Article

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