Detalhe da pesquisa
1.
A novel kinase function of a nucleoside-diphosphate-kinase homologue in Porphyromonas gingivalis is critical in subversion of host cell apoptosis by targeting heat-shock protein 27.
Cell Microbiol
; 20(5): e12825, 2018 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-29359393
2.
Inflammasome Activation in Gingival Epithelial Cells.
Methods Mol Biol
; 2459: 149-167, 2022.
Artigo
em Inglês
| MEDLINE | ID: mdl-35212963
3.
In Situ Intraepithelial Localizations of Opportunistic Pathogens, Porphyromonas gingivalis and Filifactor alocis, in Human Gingiva.
Curr Res Microb Sci
; 1: 7-17, 2020 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-34308393
4.
Porphyromonas gingivalis traffics into endoplasmic reticulum-rich-autophagosomes for successful survival in human gingival epithelial cells.
Virulence
; 9(1): 845-859, 2018 12 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-29616874
5.
NLRX1 modulates differentially NLRP3 inflammasome activation and NF-κB signaling during Fusobacterium nucleatum infection.
Microbes Infect
; 20(9-10): 615-625, 2018.
Artigo
em Inglês
| MEDLINE | ID: mdl-29024797
6.
Human Primary Epithelial Cells Acquire an Epithelial-Mesenchymal-Transition Phenotype during Long-Term Infection by the Oral Opportunistic Pathogen, Porphyromonas gingivalis.
Front Cell Infect Microbiol
; 7: 493, 2017.
Artigo
em Inglês
| MEDLINE | ID: mdl-29250491
7.
Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.
Front Cell Infect Microbiol
; 7: 291, 2017.
Artigo
em Inglês
| MEDLINE | ID: mdl-28725637
8.
Prelude to oral microbes and chronic diseases: past, present and future.
Microbes Infect
; 17(7): 473-83, 2015 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-25813714
9.
Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase.
Microbes Infect
; 17(5): 369-77, 2015 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-25828169