Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
Park, Eunjoo; Na, Hee Sam; Song, Yu-Ri; Shin, Seong Yeol; Kim, You-Me; Chung, Jin.
; 82(1): 112-23, 2014 Jan.
Artigo em Inglês | MEDLINE | ID: mdl-24126516
Porphyromonas gingivalis triggers NLRP3-mediated inflammasome activation in macrophages in a bacterial gingipains-independent manner.
Inflammasome Activation Triggers Caspase-1-Mediated Cleavage of cGAS to Regulate Responses to DNA Virus Infection.
The therapeutic potential of regulatory T lymphocytes in periodontitis: A systematic review.
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Inflammasome activation by Campylobacter jejuni.
Porphyromonas gingivalis induces the production of interleukin-31 by human mast cells, resulting in dysfunction of the gingival epithelial barrier.
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T cells instruct myeloid cells to produce inflammasome-independent IL-1ß and cause autoimmunity.
Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis.