Role of humoral mechanisms in etiology of lichen planus.
Popovska, Mirjana; Grchevska, Ladislava; Atanasovska-Stojanovska, Aneta; Kapushevska, Biljana; Muratovska, Ilijana; Minovska, Ana; Radojkova-Nikolovska, Vera; Mitik, Kristina.
Pril (Makedon Akad Nauk Umet Odd Med Nauki)
; 35(3): 185-94, 2014.
Artigo em Inglês | MEDLINE | ID: mdl-25754509
Active inflammatory biomarkers in oral lichen planus.
Histamine metabolism and transport are deranged in human keratinocytes in oral lichen planus.
The association and potentially destructive role of Th9/IL-9 is synergistic with Th17 cells by elevating MMP9 production in local lesions of oral lichen planus.
Cys-X-Cys ligand 9 might be an immunological factor in the pathogenesis of oral submucous fibrosis and its concomitant oral lichenoid lesion.
Reticular and erosive oral lichen planus have a distinct metabolomic profile: A preliminary study using gas chromatography-mass spectrometry.
Differential metallothionein expression in oral lichen planus and amalgam-associated oral lichenoid lesions.
Circulating exosomes regulate T-cell-mediated inflammatory response in oral lichen planus.
[Expression and significance of interleukin-35 in lesion tissue of oral lichen planus patients].
Lichen planus: molecular pathway and clinical implications in oral disorders.
Increased expression of p16 in both oral and genital lichen planus.