Complement Is Required for Microbe-Driven Induction of Th17 and Periodontitis.

Wang, Hui; Ideguchi, Hidetaka; Kajikawa, Tetsuhiro; Mastellos, Dimitrios C; Lambris, John D; Hajishengallis, George

Wang, Hui; Ideguchi, Hidetaka; Kajikawa, Tetsuhiro; Mastellos, Dimitrios C; Lambris, John D; Hajishengallis, George.

Afiliação

Wang H; Department of Basic and Translational Sciences, Penn Dental Medicine, University of Pennsylvania, Philadelphia, PA.
Ideguchi H; Department of Basic and Translational Sciences, Penn Dental Medicine, University of Pennsylvania, Philadelphia, PA.
Kajikawa T; Department of Pathophysiology-Periodontal Science, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama, Japan.
Mastellos DC; Department of Basic and Translational Sciences, Penn Dental Medicine, University of Pennsylvania, Philadelphia, PA.
Lambris JD; Department of Periodontology and Endodontology, Tohoku University Graduate School of Dentistry, Sendai, Japan.
Hajishengallis G; Division of Biodiagnostic Science and Technologies, The Institute of Nuclear & Radiological Sciences & Technology, Energy & Safety, National Center for Scientific Research "Demokritos," Athens, Greece; and.

J Immunol ; 209(7): 1370-1378, 2022 10 01.

Article em En | MEDLINE | ID: mdl-36028293

Assuntos

Perda do Osso Alveolar; Periodontite; Animais; Antibacterianos; Complemento C3; Humanos; Interleucina-17; Interleucina-23; Interleucina-6/metabolismo; Camundongos; Receptores de Interleucina-6; Células Th17

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Periodontite / Perda do Osso Alveolar Tipo de estudo: Clinical_trials / Prognostic_studies Limite: Animals / Humans Idioma: En Revista: J Immunol Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Panamá

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