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Cell ; 159(2): 333-45, 2014 Oct 09.
Artículo en Inglés | MEDLINE | ID: mdl-25284152

RESUMEN

In the thymus, high-affinity, self-reactive thymocytes are eliminated from the pool of developing T cells, generating central tolerance. Here, we investigate how developing T cells measure self-antigen affinity. We show that very few CD4 or CD8 coreceptor molecules are coupled with the signal-initiating kinase, Lck. To initiate signaling, an antigen-engaged T cell receptor (TCR) scans multiple coreceptor molecules to find one that is coupled to Lck; this is the first and rate-limiting step in a kinetic proofreading chain of events that eventually leads to TCR triggering and negative selection. MHCII-restricted TCRs require a shorter antigen dwell time (0.2 s) to initiate negative selection compared to MHCI-restricted TCRs (0.9 s) because more CD4 coreceptors are Lck-loaded compared to CD8. We generated a model (Lck come&stay/signal duration) that accurately predicts the observed differences in antigen dwell-time thresholds used by MHCI- and MHCII-restricted thymocytes to initiate negative selection and generate self-tolerance.


Asunto(s)
Autoantígenos/inmunología , Tolerancia Inmunológica , Receptores de Antígenos de Linfocitos T/inmunología , Animales , Antígenos de Histocompatibilidad Clase I/inmunología , Antígenos de Histocompatibilidad Clase II/inmunología , Cinética , Proteína Tirosina Quinasa p56(lck) Específica de Linfocito/metabolismo , Cadenas de Markov , Ratones Endogámicos C57BL , Receptores de Antígenos de Linfocitos T/metabolismo , Timocitos/citología , Timocitos/inmunología
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