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J Virol ; 71(2): 1718-25, 1997 Feb.
Artículo en Inglés | MEDLINE | ID: mdl-8995707

RESUMEN

Replication of the human immunodeficiency virus type 1 (HIV-1) is inhibited by interferons (IFNs), and the IFN-inducible protein kinase PKR is thought to mediate this effect by regulating protein synthesis. Here we report that ectopic expression of dominant negative PKR mutants in Jurkat cells induces HIV-1 replication. Specifically, expression of CD4 is upregulated by the PKR mutants, and this correlates with an induction of HIV-1 binding and proviral DNA synthesis upon HIV-1 infection. Moreover, activation of NF-kappaB was induced by an RNA binding-defective mutant of PKR. Thus, it appears that PKR, in addition to translational control, is involved in HIV-1 replication by modulating virus binding through the regulation of CD4 expression and virus gene expression through the activation of NF-kappaB.


Asunto(s)
Antígenos CD4/genética , Regulación Viral de la Expresión Génica , Infecciones por VIH/virología , VIH-1/fisiología , Proteínas Serina-Treonina Quinasas/genética , Replicación Viral , Técnicas de Transferencia de Gen , Humanos , Interferones/farmacología , Células Jurkat , Proteínas Serina-Treonina Quinasas/agonistas , eIF-2 Quinasa
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