Search details
1.
Loss of Tankyrase-mediated destruction of 3BP2 is the underlying pathogenic mechanism of cherubism.
Cell
; 147(6): 1324-39, 2011 Dec 09.
Article
in English
| MEDLINE | ID: mdl-22153076
2.
SMARCA4 mutation causes human otosclerosis and a similar phenotype in mice.
J Med Genet
; 61(2): 117-124, 2024 Jan 19.
Article
in English
| MEDLINE | ID: mdl-37399313
3.
Competitive blocking of LRP4-sclerostin binding interface strongly promotes bone anabolic functions.
Cell Mol Life Sci
; 79(2): 113, 2022 Jan 31.
Article
in English
| MEDLINE | ID: mdl-35099616
4.
A dual-specific macrophage colony-stimulating factor antagonist of c-FMS and αvß3 integrin for osteoporosis therapy.
PLoS Biol
; 16(8): e2002979, 2018 08.
Article
in English
| MEDLINE | ID: mdl-30142160
5.
Unraveling the transcriptional regulation of TWIST1 in limb development.
PLoS Genet
; 14(10): e1007738, 2018 10.
Article
in English
| MEDLINE | ID: mdl-30372441
6.
Positive Outcomes of Denosumab Treatment in 2 Patients With Cherubism.
J Oral Maxillofac Surg
; 78(12): 2226-2234, 2020 12.
Article
in English
| MEDLINE | ID: mdl-32649899
7.
Perturbed bone composition and integrity with disorganized osteoblast function in zinc receptor/Gpr39-deficient mice.
FASEB J
; 32(5): 2507-2518, 2018 05.
Article
in English
| MEDLINE | ID: mdl-29295862
8.
Dual-specificity tyrosine phosphorylation-regulated kinase 2 regulates osteoclast fusion in a cell heterotypic manner.
J Cell Physiol
; 233(1): 617-629, 2018 Jan.
Article
in English
| MEDLINE | ID: mdl-28332708
9.
Engineering a monomeric variant of macrophage colony-stimulating factor (M-CSF) that antagonizes the c-FMS receptor.
Biochem J
; 474(15): 2601-2617, 2017 07 20.
Article
in English
| MEDLINE | ID: mdl-28655719
10.
Combinatorial and Computational Approaches to Identify Interactions of Macrophage Colony-stimulating Factor (M-CSF) and Its Receptor c-FMS.
J Biol Chem
; 290(43): 26180-93, 2015 Oct 23.
Article
in English
| MEDLINE | ID: mdl-26359491
11.
Specific inflammatory osteoclast precursors induced during chronic inflammation give rise to highly active osteoclasts associated with inflammatory bone loss.
Bone Res
; 10(1): 36, 2022 Apr 08.
Article
in English
| MEDLINE | ID: mdl-35396510
12.
Reconstruction of the ovary microenvironment utilizing macroporous scaffold with affinity-bound growth factors.
Biomaterials
; 205: 11-22, 2019 06.
Article
in English
| MEDLINE | ID: mdl-30901634
13.
How cellular Zn2+ signaling drives physiological functions.
Cell Calcium
; 75: 53-63, 2018 11.
Article
in English
| MEDLINE | ID: mdl-30145429
14.
Schlafen2 mutation in mice causes an osteopetrotic phenotype due to a decrease in the number of osteoclast progenitors.
Sci Rep
; 8(1): 13005, 2018 08 29.
Article
in English
| MEDLINE | ID: mdl-30158544
15.
Editorial: Developmental Biology and Regulation of Osteoclasts.
Front Cell Dev Biol
; 9: 769320, 2021.
Article
in English
| MEDLINE | ID: mdl-34746157
16.
Osteoclast fusion is initiated by a small subset of RANKL-stimulated monocyte progenitors, which can fuse to RANKL-unstimulated progenitors.
Bone
; 79: 21-8, 2015 Oct.
Article
in English
| MEDLINE | ID: mdl-26008608
17.
3BP2-deficient mice are osteoporotic with impaired osteoblast and osteoclast functions.
J Clin Invest
; 121(8): 3244-57, 2011 Aug.
Article
in English
| MEDLINE | ID: mdl-21765218
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