Mutation in pre-mRNA adenosine deaminase markedly attenuates neuronal tolerance to O2 deprivation in Drosophila melanogaster.

Ma, E; Gu, X Q; Wu, X; Xu, T; Haddad, G G

Ma, E; Gu, X Q; Wu, X; Xu, T; Haddad, G G.

Affiliation

Ma E; Department of Pediatrics, Section of Respiratory Medicine, Yale University School of Medicine, 333 Cedar Street, New Haven, Connecticut 06510, USA.

J Clin Invest ; 107(6): 685-93, 2001 Mar.

Article in En | MEDLINE | ID: mdl-11254668

Subject(s)

Adenosine Deaminase/genetics; Drosophila melanogaster/genetics; Drosophila melanogaster/metabolism; Mutation; Oxygen/metabolism; Amino Acid Sequence; Animals; Animals, Genetically Modified; Base Sequence; Chromosome Mapping; DNA Primers/genetics; Disease Models, Animal; Female; Genes, Insect; Genetic Complementation Test; Humans; Hypoxia/genetics; Hypoxia/physiopathology; In Situ Hybridization; Male; Molecular Sequence Data; Neurons/metabolism; RNA-Binding Proteins; Reverse Transcriptase Polymerase Chain Reaction; Sequence Homology, Amino Acid

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Oxygen / Adenosine Deaminase / Drosophila melanogaster / Mutation Limits: Animals / Female / Humans / Male Language: En Journal: J Clin Invest Year: 2001 Type: Article Affiliation country: United States

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