Differential involvement of initiator caspases in apoptotic volume decrease and potassium efflux during Fas- and UV-induced cell death.

Vu, C C; Bortner, C D; Cidlowski, J A

Vu, C C; Bortner, C D; Cidlowski, J A.

Affiliation

Vu CC; Laboratory of Signal Transduction, NIEHS, National Institutes of Health, Research Triangle Park, North Carolina 27709, USA.

J Biol Chem ; 276(40): 37602-11, 2001 Oct 05.

Article in En | MEDLINE | ID: mdl-11431480

Subject(s)

Apoptosis/physiology; Caspases/physiology; Potassium/metabolism; fas Receptor/pharmacology; Amino Acid Chloromethyl Ketones/pharmacology; Apoptosis/radiation effects; Biological Transport/drug effects; Caspase 8; Caspase 9; Caspase Inhibitors; Cell Size/drug effects; Cell Size/physiology; Cysteine Proteinase Inhibitors/pharmacology; Enzyme Activation; Humans; Jurkat Cells; Oligopeptides/pharmacology; Ultraviolet Rays

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Collection: 01-internacional Database: MEDLINE Main subject: Potassium / Apoptosis / Fas Receptor / Caspases Limits: Humans Language: En Journal: J Biol Chem Year: 2001 Type: Article Affiliation country: United States

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