Sevoflurane depresses glutamatergic neurotransmission to brainstem inspiratory premotor neurons but not postsynaptic receptor function in a decerebrate dog model.

Stucke, Astrid G; Zuperku, Edward J; Tonkovic-Capin, Viseslav; Krolo, Mirko; Hopp, Francis A; Kampine, John P; Stuth, Eckehard A E

Stucke, Astrid G; Zuperku, Edward J; Tonkovic-Capin, Viseslav; Krolo, Mirko; Hopp, Francis A; Kampine, John P; Stuth, Eckehard A E.

Affiliation

Stucke AG; Department of Anesthesiology, Medical College of Wisconsin, Children's Hospital of Wisconsin, Milwaukee, USA.

Anesthesiology ; 103(1): 50-6, 2005 Jul.

Article in En | MEDLINE | ID: mdl-15983456

Subject(s)

Brain Stem/drug effects; Decerebrate State/physiopathology; Inhalation/drug effects; Methyl Ethers/pharmacology; Receptors, Glutamate/physiology; Animals; Brain Stem/physiology; Dogs; Dose-Response Relationship, Drug; Inhalation/physiology; Motor Neurons/drug effects; Motor Neurons/physiology; N-Methylaspartate/pharmacology; Neurons/drug effects; Neurons/physiology; Sevoflurane; Synapses/drug effects; Synapses/physiology; Synaptic Transmission/drug effects; Synaptic Transmission/physiology

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Collection: 01-internacional Database: MEDLINE Main subject: Brain Stem / Inhalation / Receptors, Glutamate / Decerebrate State / Methyl Ethers Type of study: Prognostic_studies Limits: Animals Language: En Journal: Anesthesiology Year: 2005 Type: Article Affiliation country: United States

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