TNFR1- and TNFR2-mediated signaling pathways in human kidney are cell type-specific and differentially contribute to renal injury.

Al-Lamki, Rafia S; Wang, Jun; Vandenabeele, Peter; Bradley, J Andrew; Thiru, Sathia; Luo, Dianghong; Min, Wang; Pober, Jordan S; Bradley, John R

Al-Lamki, Rafia S; Wang, Jun; Vandenabeele, Peter; Bradley, J Andrew; Thiru, Sathia; Luo, Dianghong; Min, Wang; Pober, Jordan S; Bradley, John R.

Affiliation

Al-Lamki RS; Department of Medicine, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.

FASEB J ; 19(12): 1637-45, 2005 Oct.

Article in En | MEDLINE | ID: mdl-16195372

Subject(s)

Kidney Diseases/pathology; Kidney/injuries; Kidney/metabolism; Receptors, Tumor Necrosis Factor, Type II/physiology; Receptors, Tumor Necrosis Factor, Type I/physiology; Signal Transduction; Apoptosis; Biological Assay; Biopsy; Cell Proliferation; Down-Regulation; Enzyme Activation; Humans; In Situ Hybridization; In Situ Nick-End Labeling; Kidney Neoplasms/pathology; Kidney Transplantation; Kidney Tubules/cytology; Kidney Tubules/metabolism; MAP Kinase Kinase Kinase 5/biosynthesis; Microscopy, Confocal; Models, Biological; Organ Culture Techniques; Phosphorylation; Protein-Tyrosine Kinases/metabolism; Receptors, Tumor Necrosis Factor, Type I/metabolism; Receptors, Tumor Necrosis Factor, Type II/metabolism; Threonine/chemistry; Up-Regulation

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Collection: 01-internacional Database: MEDLINE Main subject: Signal Transduction / Receptors, Tumor Necrosis Factor, Type I / Receptors, Tumor Necrosis Factor, Type II / Kidney / Kidney Diseases Type of study: Prognostic_studies Limits: Humans Language: En Journal: FASEB J Journal subject: BIOLOGIA / FISIOLOGIA Year: 2005 Type: Article Affiliation country: United kingdom

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