The Salmonella SPI2 effector SseI mediates long-term systemic infection by modulating host cell migration.

McLaughlin, Laura M; Govoni, Gregory R; Gerke, Christiane; Gopinath, Smita; Peng, Kaitian; Laidlaw, Grace; Chien, Yueh-Hsiu; Jeong, Ha-Won; Li, Zhigang; Brown, Matthew D; Sacks, David B; Monack, Denise

McLaughlin, Laura M; Govoni, Gregory R; Gerke, Christiane; Gopinath, Smita; Peng, Kaitian; Laidlaw, Grace; Chien, Yueh-Hsiu; Jeong, Ha-Won; Li, Zhigang; Brown, Matthew D; Sacks, David B; Monack, Denise.

Affiliation

McLaughlin LM; Department of Microbiology and Immunology, Stanford University Medical Center, Stanford, California, United States of America.

PLoS Pathog ; 5(11): e1000671, 2009 Nov.

Article in En | MEDLINE | ID: mdl-19956712

Subject(s)

Bacterial Proteins/physiology; Cell Movement; Host-Pathogen Interactions; Membrane Proteins/physiology; Salmonella Infections/etiology; Salmonella enterica/pathogenicity; Animals; Dendritic Cells/microbiology; Dendritic Cells/physiology; Macrophages/microbiology; Macrophages/physiology; Mice; Spleen/immunology; Time Factors

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Salmonella Infections / Bacterial Proteins / Cell Movement / Salmonella enterica / Host-Pathogen Interactions / Membrane Proteins Limits: Animals Language: En Journal: PLoS Pathog Year: 2009 Type: Article Affiliation country: United States

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