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Loss of protein tyrosine phosphatase 1B increases IGF-I receptor tyrosine phosphorylation but does not rescue retinal defects in IRS2-deficient mice.
Arroba, Ana I; Revuelta-Cervantes, Jesús; Menes, Lorena; González-Rodríguez, Águeda; Pardo, Virginia; de la Villa, Pedro; Burks, Deborah J; Valverde, Ángela M.
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  • Arroba AI; Instituto de Investigaciones Biomédicas Alberto Sols (CSIC/UAM), Madrid, Spain.
Invest Ophthalmol Vis Sci ; 54(6): 4215-25, 2013 Jun 19.
Article in En | MEDLINE | ID: mdl-23702782
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Retinal Diseases / Insulin-Like Growth Factor I / Receptor, IGF Type 1 / Receptor Protein-Tyrosine Kinases / Protein Tyrosine Phosphatase, Non-Receptor Type 1 / Insulin Receptor Substrate Proteins Type of study: Prognostic_studies Limits: Animals Language: En Journal: Invest Ophthalmol Vis Sci Year: 2013 Type: Article Affiliation country: Spain
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Retinal Diseases / Insulin-Like Growth Factor I / Receptor, IGF Type 1 / Receptor Protein-Tyrosine Kinases / Protein Tyrosine Phosphatase, Non-Receptor Type 1 / Insulin Receptor Substrate Proteins Type of study: Prognostic_studies Limits: Animals Language: En Journal: Invest Ophthalmol Vis Sci Year: 2013 Type: Article Affiliation country: Spain