Alternative splice variants of AID are not stoichiometrically present at the protein level in chronic lymphocytic leukemia.

Rebhandl, Stefan; Huemer, Michael; Zaborsky, Nadja; Gassner, Franz Josef; Catakovic, Kemal; Felder, Thomas Klaus; Greil, Richard; Geisberger, Roland

Rebhandl, Stefan; Huemer, Michael; Zaborsky, Nadja; Gassner, Franz Josef; Catakovic, Kemal; Felder, Thomas Klaus; Greil, Richard; Geisberger, Roland.

Affiliation

Rebhandl S; Laboratory for Immunological and Molecular Cancer Research, Department of Internal Medicine III with Hematology, Medical Oncology, Hemostaseology, Infectious Diseases, Rheumatology, Oncologic Center, Paracelsus Medical University, Salzburg, Austria.

Eur J Immunol ; 44(7): 2175-87, 2014 Jul.

Article in En | MEDLINE | ID: mdl-24668151

Subject(s)

Alternative Splicing; Cytidine Deaminase/genetics; Leukemia, Lymphocytic, Chronic, B-Cell/enzymology; Animals; Cytidine Deaminase/analysis; HEK293 Cells; Humans; Mice, Inbred C57BL; Real-Time Polymerase Chain Reaction

Key words

Activation-induced deaminase (AID); Alternative splicing; Chronic lymphocytic leukemia (CLL)

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Leukemia, Lymphocytic, Chronic, B-Cell / Alternative Splicing / Cytidine Deaminase Limits: Animals / Humans Language: En Journal: Eur J Immunol Year: 2014 Type: Article Affiliation country: Austria

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