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Signaling by FGF Receptor 2, Not FGF Receptor 1, Regulates Myelin Thickness through Activation of ERK1/2-MAPK, Which Promotes mTORC1 Activity in an Akt-Independent Manner.
Furusho, Miki; Ishii, Akihiro; Bansal, Rashmi.
Affiliation
  • Furusho M; Department of Neuroscience, University of Connecticut Medical School, Farmington, Connecticut 06030.
  • Ishii A; Department of Neuroscience, University of Connecticut Medical School, Farmington, Connecticut 06030.
  • Bansal R; Department of Neuroscience, University of Connecticut Medical School, Farmington, Connecticut 06030 bansal@neuron.uchc.edu.
J Neurosci ; 37(11): 2931-2946, 2017 03 15.
Article in En | MEDLINE | ID: mdl-28193689
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: MAP Kinase Signaling System / Multiprotein Complexes / Proto-Oncogene Proteins c-akt / Receptor, Fibroblast Growth Factor, Type 2 / TOR Serine-Threonine Kinases / Myelin Sheath Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neurosci Year: 2017 Type: Article
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: MAP Kinase Signaling System / Multiprotein Complexes / Proto-Oncogene Proteins c-akt / Receptor, Fibroblast Growth Factor, Type 2 / TOR Serine-Threonine Kinases / Myelin Sheath Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neurosci Year: 2017 Type: Article