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Hypermetabolic macrophages in rheumatoid arthritis and coronary artery disease due to glycogen synthase kinase 3b inactivation.
Zeisbrich, Markus; Yanes, Rolando E; Zhang, Hui; Watanabe, Ryu; Li, Yinyin; Brosig, Lukas; Hong, Jison; Wallis, Barbara B; Giacomini, John C; Assimes, Themistocles L; Goronzy, Jörg J; Weyand, Cornelia M.
Afiliación
  • Zeisbrich M; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Yanes RE; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Zhang H; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Watanabe R; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Li Y; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Brosig L; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Hong J; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Wallis BB; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Giacomini JC; Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California, USA.
  • Assimes TL; Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California, USA.
  • Goronzy JJ; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
  • Weyand CM; Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA.
Ann Rheum Dis ; 77(7): 1053-1062, 2018 07.
Article en En | MEDLINE | ID: mdl-29431119
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Artritis Reumatoide / Sinovitis / Enfermedad de la Arteria Coronaria / Glucógeno Sintasa Quinasas / Investigación Biomédica Traslacional / Macrófagos Tipo de estudio: Etiology_studies / Observational_studies / Risk_factors_studies Límite: Female / Humans / Male / Middle aged Idioma: En Revista: Ann Rheum Dis Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Artritis Reumatoide / Sinovitis / Enfermedad de la Arteria Coronaria / Glucógeno Sintasa Quinasas / Investigación Biomédica Traslacional / Macrófagos Tipo de estudio: Etiology_studies / Observational_studies / Risk_factors_studies Límite: Female / Humans / Male / Middle aged Idioma: En Revista: Ann Rheum Dis Año: 2018 Tipo del documento: Article País de afiliación: Estados Unidos