Pathogenic Function of Herpesvirus Entry Mediator in Experimental Autoimmune Uveitis by Induction of Th1- and Th17-Type T Cell Responses.

Sakoda, Yukimi; Nagai, Tomohiko; Murata, Sizuka; Mizuno, Yukari; Kurosawa, Hiromi; Shoda, Hiromi; Morishige, Naoyuki; Yanai, Ryoji; Sonoda, Koh-Hei; Tamada, Koji

Sakoda, Yukimi; Nagai, Tomohiko; Murata, Sizuka; Mizuno, Yukari; Kurosawa, Hiromi; Shoda, Hiromi; Morishige, Naoyuki; Yanai, Ryoji; Sonoda, Koh-Hei; Tamada, Koji.

Afiliación

Sakoda Y; Department of Immunology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan; and.
Nagai T; Department of Immunology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan; and Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Murata S; Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Mizuno Y; Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Kurosawa H; Department of Immunology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan; and.
Shoda H; Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Morishige N; Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Yanai R; Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Sonoda KH; Department of Ophthalmology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan.
Tamada K; Department of Immunology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi 755-8505, Japan; and ktamada@yamaguchi-u.ac.jp.

J Immunol ; 196(7): 2947-54, 2016 Apr 01.

Article en En | MEDLINE | ID: mdl-26912321

Asunto(s)

Enfermedades Autoinmunes/genética; Enfermedades Autoinmunes/inmunología; Miembro 14 de Receptores del Factor de Necrosis Tumoral/genética; Células TH1/inmunología; Células Th17/inmunología; Uveítis/genética; Uveítis/inmunología; Animales; Anticuerpos Monoclonales/farmacología; Enfermedades Autoinmunes/tratamiento farmacológico; Enfermedades Autoinmunes/metabolismo; Diferenciación Celular/genética; Citocinas/metabolismo; Modelos Animales de Enfermedad; Progresión de la Enfermedad; Ligandos; Activación de Linfocitos/inmunología; Ratones; Ratones Noqueados; Receptores Inmunológicos/metabolismo; Miembro 14 de Receptores del Factor de Necrosis Tumoral/antagonistas & inhibidores; Miembro 14 de Receptores del Factor de Necrosis Tumoral/metabolismo; Transducción de Señal; Células TH1/citología; Células TH1/metabolismo; Células Th17/citología; Células Th17/metabolismo; Miembro 14 de la Superfamilia de Ligandos de Factores de Necrosis Tumoral/metabolismo; Uveítis/tratamiento farmacológico; Uveítis/metabolismo

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Enfermedades Autoinmunes / Uveítis / Células TH1 / Miembro 14 de Receptores del Factor de Necrosis Tumoral / Células Th17 Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: J Immunol Año: 2016 Tipo del documento: Article

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