Your browser doesn't support javascript.
loading
Study of an extended family with CTLA-4 deficiency suggests a CD28/CTLA-4 independent mechanism responsible for differences in disease manifestations and severity.
Hou, Tie Zheng; Olbrich, Peter; Soto, Jose Manuel Lucena; Sanchez, Berta; Moreno, Paula Sanchez; Borte, Stephan; Stauss, Hans J; Burns, Siobhan O; Walker, Lucy S K; Pan-Hammarström, Qiang; Hammarström, Lennart; Sansom, David M; Neth, Olaf.
Afiliación
  • Hou TZ; University College London Institute of Immunity and Transplantation, School of Life and Medical Sciences, Royal Free Hospital, Rowland Hill Street, London, NW3 2PF, UK.
  • Olbrich P; Seccion de Infectología e Inmunopatología, Unidad de Pediatria, Hospital Virgen del Rocío, Sevilla, Instituto de Biomedicina de Sevilla (IBiS), Spain.
  • Soto JML; Unidad de Inmunología, Hospital Universitario Virgen del Rocío, Sevilla, Spain.
  • Sanchez B; Unidad de Inmunología, Hospital Universitario Virgen del Rocío, Sevilla, Spain.
  • Moreno PS; Seccion de Infectología e Inmunopatología, Unidad de Pediatria, Hospital Virgen del Rocío, Sevilla, Instituto de Biomedicina de Sevilla (IBiS), Spain.
  • Borte S; Division of Clinical Immunology and Transfusion Medicine, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital Huddinge, Stockholm, Sweden.; Translational Centre for Regenerative Medicine (TRM), University of Leipzig, Leipzig, Germany; ImmunoDeficiencyCenter Leipz
  • Stauss HJ; University College London Institute of Immunity and Transplantation, School of Life and Medical Sciences, Royal Free Hospital, Rowland Hill Street, London, NW3 2PF, UK.
  • Burns SO; University College London Institute of Immunity and Transplantation, School of Life and Medical Sciences, Royal Free Hospital, Rowland Hill Street, London, NW3 2PF, UK.
  • Walker LSK; University College London Institute of Immunity and Transplantation, School of Life and Medical Sciences, Royal Free Hospital, Rowland Hill Street, London, NW3 2PF, UK.
  • Pan-Hammarström Q; Division of Clinical Immunology and Transfusion Medicine, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital Huddinge, Stockholm, Sweden.
  • Hammarström L; Division of Clinical Immunology and Transfusion Medicine, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital Huddinge, Stockholm, Sweden.
  • Sansom DM; University College London Institute of Immunity and Transplantation, School of Life and Medical Sciences, Royal Free Hospital, Rowland Hill Street, London, NW3 2PF, UK. Electronic address: d.sansom@ucl.ac.uk.
  • Neth O; Seccion de Infectología e Inmunopatología, Unidad de Pediatria, Hospital Virgen del Rocío, Sevilla, Instituto de Biomedicina de Sevilla (IBiS), Spain.
Clin Immunol ; 188: 94-102, 2018 03.
Article en En | MEDLINE | ID: mdl-29305966
Asunto(s)
Palabras clave
Texto completo
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Activación de Linfocitos / Linfocitos T / Antígenos CD28 / Antígeno CTLA-4 Límite: Female / Humans / Male Idioma: En Revista: Clin Immunol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2018 Tipo del documento: Article País de afiliación: Reino Unido
Texto completo
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Activación de Linfocitos / Linfocitos T / Antígenos CD28 / Antígeno CTLA-4 Límite: Female / Humans / Male Idioma: En Revista: Clin Immunol Asunto de la revista: ALERGIA E IMUNOLOGIA Año: 2018 Tipo del documento: Article País de afiliación: Reino Unido