Deficiency of Anoctamin 5/TMEM16E causes nuclear positioning defect and impairs Ca<sup>2+</sup> signaling of differentiated C2C12 myotubes.

Phuong, Tam Thi Thanh; An, Jieun; Park, Sun Hwa; Kim, Ami; Choi, Hyun Bin; Kang, Tong Mook

Deficiency of Anoctamin 5/TMEM16E causes nuclear positioning defect and impairs Ca²⁺ signaling of differentiated C2C12 myotubes.

Phuong, Tam Thi Thanh; An, Jieun; Park, Sun Hwa; Kim, Ami; Choi, Hyun Bin; Kang, Tong Mook.

Afiliación

Phuong TTT; Department of Physiology, Sungkyunkwan University School of Medicine, Suwon 16419, Korea.
An J; Department of Physiology, Sungkyunkwan University School of Medicine, Suwon 16419, Korea.
Park SH; Department of Physiology, Sungkyunkwan University School of Medicine, Suwon 16419, Korea.
Kim A; Department of Physiology, Sungkyunkwan University School of Medicine, Suwon 16419, Korea.
Choi HB; Department of Physiology, Sungkyunkwan University School of Medicine, Suwon 16419, Korea.
Kang TM; Department of Physiology, Sungkyunkwan University School of Medicine, Suwon 16419, Korea.

Korean J Physiol Pharmacol ; 23(6): 539-547, 2019 Nov.

Article en En | MEDLINE | ID: mdl-31680776

Palabras clave

Anoctamin 5; C2C12; Excitation-contraction coupling; Kif5b; Myoblast differentiation; Nuclear positioning

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Tipo de estudio: Etiology_studies Idioma: En Revista: Korean J Physiol Pharmacol Año: 2019 Tipo del documento: Article

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