Modelling cognitive deficits in Parkinson's disease: Is CA2 a gateway for hippocampal synucleinopathy?

Cinar, Elif; Yalcin-Cakmakli, Gul; Saka, Esen; Ulusoy, Ayse; Yuruker, Sinan; Elibol, Bulent; Tel, Banu C

Cinar, Elif; Yalcin-Cakmakli, Gul; Saka, Esen; Ulusoy, Ayse; Yuruker, Sinan; Elibol, Bulent; Tel, Banu C.

Afiliación

Cinar E; Department of Pharmacology, Faculty of Pharmacy, Hacettepe University, Ankara, Turkey. Electronic address: elif.cinar@hacettepe.edu.tr.
Yalcin-Cakmakli G; Department of Neurology, Hacettepe University, Sihhiye, Ankara, Turkey.
Saka E; Department of Neurology, Hacettepe University, Sihhiye, Ankara, Turkey.
Ulusoy A; German Center for Neurodegenerative Diseases, Bonn, Germany.
Yuruker S; Usak University Faculty of Medicine, Department of Histology and Embryology, Usak, Turkey.
Elibol B; Department of Neurology, Hacettepe University, Sihhiye, Ankara, Turkey.
Tel BC; Department of Pharmacology, Faculty of Pharmacy, Hacettepe University, Ankara, Turkey.

Exp Neurol ; 330: 113357, 2020 08.

Article en En | MEDLINE | ID: mdl-32437708

Asunto(s)

Región CA2 Hipocampal/patología; Disfunción Cognitiva; Modelos Animales de Enfermedad; Enfermedad de Parkinson/patología; alfa-Sinucleína/toxicidad; Animales; Giro Dentado/patología; Femenino; Humanos; Ratas; Ratas Sprague-Dawley; Sustancia Negra/patología

Palabras clave

Adeno-associated viral vector; Alpha-synuclein; CA2 subregion; Cognitive deficits; Parkinson's disease; Synaptic dysfunction

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Modelos Animales de Enfermedad / Alfa-Sinucleína / Región CA2 Hipocampal / Disfunción Cognitiva Límite: Animals / Female / Humans Idioma: En Revista: Exp Neurol Año: 2020 Tipo del documento: Article

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