Mu opioid receptor-mediated release of endolysosome iron increases levels of mitochondrial iron, reactive oxygen species, and cell death.

Halcrow, Peter W; Kumar, Nirmal; Hao, Emily; Khan, Nabab; Meucci, Olimpia; Geiger, Jonathan D

Halcrow, Peter W; Kumar, Nirmal; Hao, Emily; Khan, Nabab; Meucci, Olimpia; Geiger, Jonathan D.

Afiliación

Halcrow PW; Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA.
Kumar N; Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA.
Hao E; Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA.
Khan N; Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA.
Meucci O; Department of Physiology and Pharmacology, Drexel University School of Medicine, Philadelphia, PA, USA.
Geiger JD; Department of Biomedical Sciences, University of North Dakota School of Medicine and Health Sciences, Grand Forks, ND, USA.

NeuroImmune Pharm Ther ; 2(1): 19-35, 2023 Mar 25.

Article en En | MEDLINE | ID: mdl-37027339

Palabras clave

cell death; deferoxamine; endolysosomes; iron; mitochondria; opioids

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Idioma: En Revista: NeuroImmune Pharm Ther Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos

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