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The frustrated host response to Legionella pneumophila is bypassed by MyD88-dependent translation of pro-inflammatory cytokines.
Asrat, Seblewongel; Dugan, Aisling S; Isberg, Ralph R.
Afiliación
  • Asrat S; Howard Hughes Medical Institute, Boston, Massachusetts, United States of America; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, United States of America; Graduate Program in Molecular Microbiology, Sackler School of Graduate Biomedical Science, Tufts University School of Medicine, Boston, Massachusetts, United States of America.
  • Dugan AS; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, United States of America.
  • Isberg RR; Howard Hughes Medical Institute, Boston, Massachusetts, United States of America; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, United States of America.
PLoS Pathog ; 10(7): e1004229, 2014 Jul.
Article en En | MEDLINE | ID: mdl-25058342
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Biosíntesis de Proteínas / Enfermedad de los Legionarios / Legionella pneumophila / Interleucina-1alfa / Interleucina-1beta / Factor 88 de Diferenciación Mieloide Límite: Animals / Humans Idioma: En Revista: PLoS Pathog Año: 2014 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Biosíntesis de Proteínas / Enfermedad de los Legionarios / Legionella pneumophila / Interleucina-1alfa / Interleucina-1beta / Factor 88 de Diferenciación Mieloide Límite: Animals / Humans Idioma: En Revista: PLoS Pathog Año: 2014 Tipo del documento: Article País de afiliación: Estados Unidos