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Arrhythmogenic Biophysical Phenotype for SCN5A Mutation S1787N Depends upon Splice Variant Background and Intracellular Acidosis.
Hu, Rou-Mu; Tan, Bi-Hua; Tester, David J; Song, Chunhua; He, Yang; Dovat, Sinisa; Peterson, Blaise Z; Ackerman, Michael J; Makielski, Jonathan C.
Afiliación
  • Hu RM; Department of Cardiology, China Meitan General Hospital, Beijing, China; Department of Medicine, Division of Cardiovascular Medicine, University of Wisconsin, Madison, WI, United States of America.
  • Tan BH; Department of Medicine, Division of Cardiovascular Medicine, University of Wisconsin, Madison, WI, United States of America; Departments of Pediatrics, and Cellular & Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, PA, United States of America.
  • Tester DJ; Departments of Medicine, Pediatrics, and Molecular Pharmacology and Experimental Therapeutics, Mayo Clinic, Rochester, MN, United States of America.
  • Song C; Departments of Pediatrics, and Cellular & Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, PA, United States of America.
  • He Y; Department of Medicine, Division of Cardiovascular Medicine, University of Wisconsin, Madison, WI, United States of America.
  • Dovat S; Departments of Pediatrics, and Cellular & Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, PA, United States of America.
  • Peterson BZ; Departments of Pediatrics, and Cellular & Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, PA, United States of America.
  • Ackerman MJ; Departments of Medicine, Pediatrics, and Molecular Pharmacology and Experimental Therapeutics, Mayo Clinic, Rochester, MN, United States of America.
  • Makielski JC; Department of Medicine, Division of Cardiovascular Medicine, University of Wisconsin, Madison, WI, United States of America.
PLoS One ; 10(4): e0124921, 2015.
Article en En | MEDLINE | ID: mdl-25923670
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Acidosis / Empalme del ARN / Canal de Sodio Activado por Voltaje NAV1.5 Tipo de estudio: Etiology_studies Límite: Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2015 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Acidosis / Empalme del ARN / Canal de Sodio Activado por Voltaje NAV1.5 Tipo de estudio: Etiology_studies Límite: Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2015 Tipo del documento: Article País de afiliación: Estados Unidos