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HPV16 and 18 genome amplification show different E4-dependence, with 16E4 enhancing E1 nuclear accumulation and replicative efficiency via its cell cycle arrest and kinase activation functions.
Egawa, Nagayasu; Wang, Qian; Griffin, Heather M; Murakami, Isao; Jackson, Deborah; Mahmood, Radma; Doorbar, John.
Afiliación
  • Egawa N; Department of Pathology, University of Cambridge, Cambridge, United Kingdom.
  • Wang Q; Francis Crick Institute, Mill Hill Laboratory, The Ridgeway, Mill Hill, London, United Kingdom.
  • Griffin HM; Francis Crick Institute, Mill Hill Laboratory, The Ridgeway, Mill Hill, London, United Kingdom.
  • Murakami I; Department of Pathology, University of Cambridge, Cambridge, United Kingdom.
  • Jackson D; Francis Crick Institute, Mill Hill Laboratory, The Ridgeway, Mill Hill, London, United Kingdom.
  • Mahmood R; Department of Pathology, University of Cambridge, Cambridge, United Kingdom.
  • Doorbar J; Francis Crick Institute, Mill Hill Laboratory, The Ridgeway, Mill Hill, London, United Kingdom.
PLoS Pathog ; 13(3): e1006282, 2017 03.
Article en En | MEDLINE | ID: mdl-28306742
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Replicación Viral / Proteínas Oncogénicas Virales / Genoma Viral / Papillomavirus Humano 16 / Puntos de Control del Ciclo Celular Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: PLoS Pathog Año: 2017 Tipo del documento: Article País de afiliación: Reino Unido
Texto completo
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Replicación Viral / Proteínas Oncogénicas Virales / Genoma Viral / Papillomavirus Humano 16 / Puntos de Control del Ciclo Celular Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: PLoS Pathog Año: 2017 Tipo del documento: Article País de afiliación: Reino Unido