The Role of N-&#945;-acetyltransferase 10 Protein in DNA Methylation and Genomic Imprinting.

Lee, Chen-Cheng; Peng, Shih-Huan; Shen, Li; Lee, Chung-Fan; Du, Ting-Huei; Kang, Ming-Lun; Xu, Guo-Liang; Upadhyay, Anup K; Cheng, Xiaodong; Yan, Yu-Ting; Zhang, Yi; Juan, Li-Jung

The Role of N-α-acetyltransferase 10 Protein in DNA Methylation and Genomic Imprinting.

Lee, Chen-Cheng; Peng, Shih-Huan; Shen, Li; Lee, Chung-Fan; Du, Ting-Huei; Kang, Ming-Lun; Xu, Guo-Liang; Upadhyay, Anup K; Cheng, Xiaodong; Yan, Yu-Ting; Zhang, Yi; Juan, Li-Jung.

Afiliación

Lee CC; Genomics Research Center, Academia Sinica, Taipei 115, Taiwan, ROC.
Peng SH; Genomics Research Center, Academia Sinica, Taipei 115, Taiwan, ROC; Institute of Molecular Medicine, National Taiwan University College of Medicine, Taipei 100, Taiwan, ROC.
Shen L; Howard Hughes Medical Institute, Harvard Medical School, Boston, MA 02115, USA.
Lee CF; Genomics Research Center, Academia Sinica, Taipei 115, Taiwan, ROC.
Du TH; Genomics Research Center, Academia Sinica, Taipei 115, Taiwan, ROC.
Kang ML; Genomics Research Center, Academia Sinica, Taipei 115, Taiwan, ROC.
Xu GL; Institute of Biochemistry and Cell Biology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200031, China.
Upadhyay AK; Department of Biochemistry, Emory University School of Medicine, Atlanta, GA 30322, USA.
Cheng X; Department of Biochemistry, Emory University School of Medicine, Atlanta, GA 30322, USA; Department of Molecular and Cellular Oncology, The University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA.
Yan YT; Institute of Biomedical Sciences, Academia Sinica, Taipei 115, Taiwan, ROC.
Zhang Y; Howard Hughes Medical Institute, Harvard Medical School, Boston, MA 02115, USA. Electronic address: yzhang@genetics.med.harvard.edu.
Juan LJ; Genomics Research Center, Academia Sinica, Taipei 115, Taiwan, ROC. Electronic address: ljjuan@gate.sinica.edu.tw.

Mol Cell ; 68(1): 89-103.e7, 2017 Oct 05.

Article en En | MEDLINE | ID: mdl-28943313

Asunto(s)

ADN (Citosina-5-)-Metiltransferasas/genética; Discapacidades del Desarrollo/genética; Epigénesis Genética; Impresión Genómica; Acetiltransferasa A N-Terminal/genética; Acetiltransferasa E N-Terminal/genética; ARN Largo no Codificante/genética; Animales; ADN/genética; ADN/metabolismo; ADN (Citosina-5-)-Metiltransferasa 1; ADN (Citosina-5-)-Metiltransferasas/metabolismo; Metilación de ADN; Discapacidades del Desarrollo/metabolismo; Discapacidades del Desarrollo/patología; Modelos Animales de Enfermedad; Embrión de Mamíferos; Femenino; Eliminación de Gen; Genes Letales; Estudio de Asociación del Genoma Completo; Masculino; Ratones; Ratones Endogámicos C57BL; Ratones Noqueados; Células Madre Embrionarias de Ratones/metabolismo; Células Madre Embrionarias de Ratones/patología; Acetiltransferasa A N-Terminal/deficiencia; Acetiltransferasa E N-Terminal/deficiencia; Unión Proteica; ARN Largo no Codificante/metabolismo; Fase S/genética

Palabras clave

DNA methylation; Dnmt1; N-α-acetyltransferase; Naa10p; Ogden syndrome; development; developmental delay; genomic imprinting

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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Discapacidades del Desarrollo / Impresión Genómica / Epigénesis Genética / ADN (Citosina-5-)-Metiltransferasas / Acetiltransferasa A N-Terminal / Acetiltransferasa E N-Terminal / ARN Largo no Codificante Límite: Animals Idioma: En Revista: Mol Cell Asunto de la revista: BIOLOGIA MOLECULAR Año: 2017 Tipo del documento: Article

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