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TPNA10168, an Nrf-2 activator, attenuates inflammatory responses independently of Nrf2 in microglial BV-2 cells: Involvement of the extracellular-signal-regulated kinase pathway.
Izumi, Yasuhiko; Tatsumoto, Ai; Horiuchi, Naoko; Arifuku, Monami; Uegomori, Momoko; Kume, Toshiaki; Koyama, Yutaka.
Afiliación
  • Izumi Y; Laboratory of Pharmacology, Kobe Pharmaceutical University, 4-19-1 Motoyamakita-machi, Higashinada-ku, Kobe, 658-8558, Japan. Electronic address: y-izumi@kobepharma-u.ac.jp.
  • Tatsumoto A; Laboratory of Pharmacology, Kobe Pharmaceutical University, 4-19-1 Motoyamakita-machi, Higashinada-ku, Kobe, 658-8558, Japan.
  • Horiuchi N; Laboratory of Pharmacology, Kobe Pharmaceutical University, 4-19-1 Motoyamakita-machi, Higashinada-ku, Kobe, 658-8558, Japan.
  • Arifuku M; Department of Pharmacology, Graduate School of Pharmaceutical Sciences, Kyoto University, 46-29 Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan.
  • Uegomori M; Laboratory of Pharmacology, Kobe Pharmaceutical University, 4-19-1 Motoyamakita-machi, Higashinada-ku, Kobe, 658-8558, Japan.
  • Kume T; Department of Applied Pharmacology, Graduate School of Medical and Pharmaceutical Science, University of Toyama, 2630 Sugitani, Toyama, 930-0194, Japan.
  • Koyama Y; Laboratory of Pharmacology, Kobe Pharmaceutical University, 4-19-1 Motoyamakita-machi, Higashinada-ku, Kobe, 658-8558, Japan.
J Pharmacol Sci ; 149(1): 1-10, 2022 May.
Article en En | MEDLINE | ID: mdl-35369899
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Quinasas MAP Reguladas por Señal Extracelular / Factor 2 Relacionado con NF-E2 Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: J Pharmacol Sci Asunto de la revista: FARMACOLOGIA Año: 2022 Tipo del documento: Article
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Texto completo: 1 Base de datos: MEDLINE Asunto principal: Quinasas MAP Reguladas por Señal Extracelular / Factor 2 Relacionado con NF-E2 Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: J Pharmacol Sci Asunto de la revista: FARMACOLOGIA Año: 2022 Tipo del documento: Article