Updates in the chronic kidney disease-mineral bone disorder show the role of osteocytic proteins, a potential mechanism of the bone-Vascular paradox, a therapeutic target, and a biomarker.

Williams, Matthew J; White, Sarah C; Joseph, Zachary; Hruska, Keith A

Williams, Matthew J; White, Sarah C; Joseph, Zachary; Hruska, Keith A.

Afiliación

Williams MJ; Division of Pediatric Nephrology, Department of Pediatrics, Washington University, Saint Louis, MO, United States.
White SC; Division of Pediatric Nephrology, Department of Pediatrics, Washington University, Saint Louis, MO, United States.
Joseph Z; Division of Pediatric Nephrology, Department of Pediatrics, Washington University, Saint Louis, MO, United States.
Hruska KA; Division of Pediatric Nephrology, Department of Pediatrics, Washington University, Saint Louis, MO, United States.

Front Physiol ; 14: 1120308, 2023.

Article en En | MEDLINE | ID: mdl-36776982

Palabras clave

chronic kidney disease; chronic kidney disease-mineral bone disorder; fibroblast growth factor 23; klotho; parathyroid hormone; renal osteodystrophy; sclerostin (SOST); vascular calcification

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Texto completo: 1 Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Front Physiol Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos

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