Detalhe da pesquisa
1.
Role of KCNK3 Dysfunction in Dasatinib-associated PAH and Endothelial Cell Dysfunction.
Am J Respir Cell Mol Biol
; 2024 Mar 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-38546978
2.
Orai1 Inhibitors as Potential Treatments for Pulmonary Arterial Hypertension.
Circ Res
; 131(9): e102-e119, 2022 10 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-36164973
3.
Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart.
J Physiol
; 601(17): 3717-3737, 2023 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-37477289
4.
Contribution of transient receptor potential canonical channels in human and experimental pulmonary arterial hypertension.
Am J Physiol Lung Cell Mol Physiol
; 325(2): L246-L261, 2023 08 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37366608
5.
SUR1 As a New Therapeutic Target for Pulmonary Arterial Hypertension.
Am J Respir Cell Mol Biol
; 66(5): 539-554, 2022 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-35175177
6.
Characterization of Kcnk3-Mutated Rat, a Novel Model of Pulmonary Hypertension.
Circ Res
; 125(7): 678-695, 2019 09 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-31347976
7.
Proteomic Analysis of KCNK3 Loss of Expression Identified Dysregulated Pathways in Pulmonary Vascular Cells.
Int J Mol Sci
; 21(19)2020 Oct 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-33036472
8.
Response to a Letter to the Editor: Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart.
J Physiol
; 602(9): 2143-2144, 2024 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-38648379
9.
T-type Ca2+ channels elicit pro-proliferative and anti-apoptotic responses through impaired PP2A/Akt1 signaling in PASMCs from patients with pulmonary arterial hypertension.
Biochim Biophys Acta Mol Cell Res
; 1864(10): 1631-1641, 2017 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-28655554
10.
Ion Channels in Pulmonary Hypertension: A Therapeutic Interest?
Int J Mol Sci
; 19(10)2018 Oct 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-30322215
11.
Orai1/STIMs modulators in pulmonary vascular diseases.
Cell Calcium
; 121: 102892, 2024 Apr 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-38735127
12.
T-type Ca²âº signalling downregulates MEK1/2 phosphorylation and cross-talk with the RAAS transcriptional response in cardiac myocytes.
J Mol Cell Cardiol
; 53(2): 291-8, 2012 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-22634504
13.
Involvement of SUR2/Kir6.1 channel in the physiopathology of pulmonary arterial hypertension.
Front Cardiovasc Med
; 9: 1066047, 2022.
Artigo
em Inglês
| MEDLINE | ID: mdl-36704469
14.
Role of Store-Operated Ca2+ Entry in the Pulmonary Vascular Remodeling Occurring in Pulmonary Arterial Hypertension.
Biomolecules
; 11(12)2021 11 27.
Artigo
em Inglês
| MEDLINE | ID: mdl-34944425
15.
Retraction notice to ICAM-1 PROMOTES THE ABNORMAL ENDOTHELIAL CELL PHENOTYPE IN CHRONIC THROMBOEMBOLIC PULMONARY HYPERTENSION.
J Heart Lung Transplant
; 40(4): 318, 2021 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-33810826
16.
Kcnk3 dysfunction exaggerates the development of pulmonary hypertension induced by left ventricular pressure overload.
Cardiovasc Res
; 117(12): 2474-2488, 2021 11 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33483721
17.
Identification of functional corticosteroid response elements involved in regulation of Cacna1g expression in cardiac myocytes.
Mol Cell Biochem
; 335(1-2): 47-51, 2010 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-19705257
18.
Implication of Potassium Channels in the Pathophysiology of Pulmonary Arterial Hypertension.
Biomolecules
; 10(9)2020 09 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-32882918
19.
Regulation of T-type Cav3.1 channels expression by synthetic glucocorticoid dexamethasone in neonatal cardiac myocytes.
Mol Cell Biochem
; 320(1-2): 173-83, 2009 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-18820838
20.
ICAM-1 promotes the abnormal endothelial cell phenotype in chronic thromboembolic pulmonary hypertension.
J Heart Lung Transplant
; 38(9): 982-996, 2019 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-31324443