Detalhe da pesquisa
1.
NTR 2.0: a rationally engineered prodrug-converting enzyme with substantially enhanced efficacy for targeted cell ablation.
Nat Methods
; 19(2): 205-215, 2022 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-35132245
2.
Conditional Creation and Rescue of Nipbl-Deficiency in Mice Reveals Multiple Determinants of Risk for Congenital Heart Defects.
PLoS Biol
; 14(9): e2000197, 2016 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-27606604
3.
Nipbl and mediator cooperatively regulate gene expression to control limb development.
PLoS Genet
; 10(9): e1004671, 2014 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-25255084
4.
Genetic enhancement of limb defects in a mouse model of Cornelia de Lange syndrome.
Am J Med Genet C Semin Med Genet
; 172(2): 146-54, 2016 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-27120109
5.
Using mouse and zebrafish models to understand the etiology of developmental defects in Cornelia de Lange Syndrome.
Am J Med Genet C Semin Med Genet
; 172(2): 138-45, 2016 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-27120001
6.
Gastrulation-stage gene expression in Nipbl+/- mouse embryos foreshadows the development of syndromic birth defects.
bioRxiv
; 2024 Feb 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-37905011
7.
Gastrulation-stage gene expression in Nipbl+/- mouse embryos foreshadows the development of syndromic birth defects.
Sci Adv
; 10(12): eadl4239, 2024 Mar 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-38507484
8.
Multiple organ system defects and transcriptional dysregulation in the Nipbl(+/-) mouse, a model of Cornelia de Lange Syndrome.
PLoS Genet
; 5(9): e1000650, 2009 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-19763162