Detalhe da pesquisa
1.
Genetic Deletion of Galectin-3 Exacerbates Age-Related Myocardial Hypertrophy and Fibrosis in Mice.
Cell Physiol Biochem
; 56(4): 353-366, 2022 Aug 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-35959709
2.
The combined action of glycoinositolphospholipid from Trypanosoma cruzi and macrophage migration inhibitory factor increases proinflammatory mediator production by cardiomyocytes and vascular endothelial cells.
Microb Pathog
; 173(Pt A): 105881, 2022 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-36379373
3.
Genetic Deletion of Galectin-3 Alters the Temporal Evolution of Macrophage Infiltration and Healing Affecting the Cardiac Remodeling and Function after Myocardial Infarction in Mice.
Am J Pathol
; 190(9): 1789-1800, 2020 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-32473918
4.
Treatment in vitro with PPARα and PPARγ ligands drives M1-to-M2 polarization of macrophages from T. cruzi-infected mice.
Biochim Biophys Acta
; 1852(5): 893-904, 2015 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-25557389
5.
Hepatic injury associated with Trypanosoma cruzi infection is attenuated by treatment with 15-deoxy-Δ12,14 prostaglandin J2.
Exp Parasitol
; 170: 100-108, 2016 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-27693222
6.
PPARγ ligand treatment inhibits cardiac inflammatory mediators induced by infection with different lethality strains of Trypanosoma cruzi.
Biochim Biophys Acta
; 1832(1): 239-48, 2013 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-22917565
7.
Fenofibrate Induces a Resolving Profile in Heart Macrophage Subsets and Attenuates Acute Chagas Myocarditis.
ACS Infect Dis
; 10(5): 1793-1807, 2024 May 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-38648355
8.
Macrophages derived from septic mice modulate nitric oxide synthase and angiogenic mediators in the heart.
J Cell Physiol
; 228(7): 1584-93, 2013 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-23335284
9.
Macrophages Mediate Healing Properties of Fenofibrate in Experimental Chagasic Cardiomyopathy.
ACS Infect Dis
; 9(2): 213-220, 2023 02 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-36661566
10.
Studies on the contribution of PPAR Gamma to tuberculosis physiopathology.
Front Cell Infect Microbiol
; 13: 1067464, 2023.
Artigo
em Inglês
| MEDLINE | ID: mdl-37187471
11.
Galectin-3 contributes to acute cardiac dysfunction and toxicity by increasing oxidative stress and fibrosis in doxorubicin-treated mice.
Int J Cardiol
; 393: 131386, 2023 12 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-37741348
12.
Benznidazole Anti-Inflammatory Effects in Murine Cardiomyocytes and Macrophages Are Mediated by Class I PI3Kδ.
Front Immunol
; 12: 782891, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-34925364
13.
Fenofibrate Increases the Population of Non-Classical Monocytes in Asymptomatic Chagas Disease Patients and Modulates Inflammatory Cytokines in PBMC.
Front Cell Infect Microbiol
; 11: 785166, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-35360222
14.
Involvement of lipids from Leishmania braziliensis promastigotes and amastigotes in macrophage activation.
Mol Immunol
; 125: 104-114, 2020 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-32659595
15.
IL-10-Dependent and -Independent Mechanisms Are Involved in the Cardiac Pathology Modulation Mediated by Fenofibrate in an Experimental Model of Chagas Heart Disease.
Front Immunol
; 11: 572178, 2020.
Artigo
em Inglês
| MEDLINE | ID: mdl-33072115
16.
IL-10/STAT3/SOCS3 Axis Is Involved in the Anti-inflammatory Effect of Benznidazole.
Front Immunol
; 10: 1267, 2019.
Artigo
em Inglês
| MEDLINE | ID: mdl-31214200
17.
Pyridinecarboxylic Acid Derivative Stimulates Pro-Angiogenic Mediators by PI3K/AKT/mTOR and Inhibits Reactive Nitrogen and Oxygen Species and NF-κB Activation Through a PPARγ-Dependent Pathway in T. cruzi-Infected Macrophages.
Front Immunol
; 10: 2955, 2019.
Artigo
em Inglês
| MEDLINE | ID: mdl-31993046
18.
Treatment with Fenofibrate plus a low dose of Benznidazole attenuates cardiac dysfunction in experimental Chagas disease.
Int J Parasitol Drugs Drug Resist
; 7(3): 378-387, 2017 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-29040909
19.
Treatment with a New Peroxisome Proliferator-Activated Receptor Gamma Agonist, Pyridinecarboxylic Acid Derivative, Increases Angiogenesis and Reduces Inflammatory Mediators in the Heart of Trypanosoma cruzi-Infected Mice.
Front Immunol
; 8: 1738, 2017.
Artigo
em Inglês
| MEDLINE | ID: mdl-29312293
20.
Low-dose benznidazole treatment results in parasite clearance and attenuates heart inflammatory reaction in an experimental model of infection with a highly virulent Trypanosoma cruzi strain.
Int J Parasitol Drugs Drug Resist
; 6(1): 12-22, 2016 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-26862474