Detalhe da pesquisa
1.
Novel Role for Cardiolipin as a Target of Therapy to Mitigate Myocardial Injury Caused by Venoarterial Extracorporeal Membrane Oxygenation.
Circulation
; 149(17): 1341-1353, 2024 Apr 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-38235580
2.
Redox Imaging Using Cardiac Myocyte-Specific Transgenic Biosensor Mice.
Circ Res
; 119(9): 1004-1016, 2016 Oct 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-27553648
3.
Proceedings of the 3rd annual Acute Cardiac Unloading and REcovery (A-CURE) symposium.
BMC Cardiovasc Disord
; 19(Suppl 2): 27, 2019 02 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-30732562
4.
Circulating Proteome Analysis Identifies Reduced Inflammation After Initiation of Hemodynamic Support with Either Veno-Arterial Extracorporeal Membrane Oxygenation or Impella in Patients with Cardiogenic Shock.
J Cardiovasc Transl Res
; 2024 Feb 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-38409476
5.
ALK1 Deficiency Impairs the Wound-Healing Process and Increases Mortality in Murine Model of Myocardial Infarction.
J Cardiovasc Transl Res
; 2023 Dec 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-38064044
6.
Loss of Bone Morphogenetic Protein-9 Reduces Survival and Increases MMP Activity After Myocardial Infarction.
JACC Basic Transl Sci
; 8(10): 1318-1330, 2023 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-38094696
7.
Transvalvular Unloading Mitigates Ventricular Injury Due to Venoarterial Extracorporeal Membrane Oxygenation in Acute Myocardial Infarction.
JACC Basic Transl Sci
; 8(7): 769-780, 2023 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-37547066
8.
Myocardial Injury Promotes Matrix Metalloproteinase-9 Activity in the Renal Cortex in Preclinical Models of Acute Myocardial Infarction.
J Cardiovasc Transl Res
; 15(2): 207-216, 2022 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-33782857
9.
Systemic Inflammatory Burden Correlates with Severity and Predicts Outcomes in Patients with Cardiogenic Shock Supported by a Percutaneous Mechanical Assist Device.
J Cardiovasc Transl Res
; 14(3): 476-483, 2021 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-33078375
10.
Transvalvular Ventricular Unloading Before Reperfusion in Acute Myocardial Infarction.
J Am Coll Cardiol
; 76(6): 684-699, 2020 08 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-32762903
11.
Mechanical Left Ventricular Unloading to Reduce Infarct Size During Acute Myocardial Infarction: Insight from Preclinical and Clinical Studies.
J Cardiovasc Transl Res
; 12(2): 87-94, 2019 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-31016553
12.
Left Ventricular Unloading Increases the Coronary Collateral Flow Index Before Reperfusion and Reduces Infarct Size in a Swine Model of Acute Myocardial Infarction.
J Am Heart Assoc
; 8(22): e013586, 2019 11 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-31698989
13.
Mechanically Regulating Cardiac Preload to Maximize Left Ventricular Unloading With a Transvalvular Microaxial Flow Pump.
Circ Heart Fail
; 17(4): e011330, 2024 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-38626066
14.
Transgenic Organisms Meet Redox Bioimaging: One Step Closer to Physiology.
Antioxid Redox Signal
; 29(6): 603-612, 2018 08 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-29320870
15.
Correction to: Systemic Inflammatory Burden Correlates with Severity and Predicts Outcomes in Patients with Cardiogenic Shock Supported by a Percutaneous Mechanical Assist Device.
J Cardiovasc Transl Res
; 16(3): 754, 2023 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-36417125
16.
Loss of PHD3 in myeloid cells dampens the inflammatory response and fibrosis after hind-limb ischemia.
Cell Death Dis
; 8(8): e2976, 2017 08 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-28796258
17.
PHD2 Is a Regulator for Glycolytic Reprogramming in Macrophages.
Mol Cell Biol
; 37(1)2017 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-27795296
18.
Prolyl-4-hydroxylase domain 3 (PHD3) is a critical terminator for cell survival of macrophages under stress conditions.
J Leukoc Biol
; 96(3): 365-75, 2014 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-24626957